本文报道了我国首例家族性脂蛋白脂酶缺乏症。
The first case of familial lipoprotein lipase (LPL) deficiency in China is re-ported.
目的探讨脂蛋白脂酶（LPL）基因多态性与冠心病的关系。
Objective To explore the association of LPL gene polymorphisms with CHD.
目的：使用纯化的脂蛋白脂酶，用体外法研究锰对脂蛋白脂酶活性的影响。
Objective To study the effect of tea polyphenols on mesangial cell ingesting lipoprotein a(LPa).
结论肾病综合征脂蛋白脂酶和肝脂酶活性降低为高三酰甘油血症的病因之一。
Conclusions Reduced activity of lipoprotein lipase and hepatic lipase is one of causes leading to hypertriglyceridemia in nephrotic syndrome.
脂蛋白脂酶可能通过参与子痫前期高血脂异常代谢对子痫前期发病起着重要作用。
LPL possibly has a significant role in the pathogenesis of the case through participating in lipids abnormal metabolism.
血管壁中巨噬细胞产生脂蛋白脂酶，使脂质积聚于动脉壁内，促进动脉粥样硬化进展。
Macrophages constitutively synthesize LPL, which may contribute to lipid accumulation within the arterial wall, promoting the atherogenic process.
脂蛋白脂酶（LPL）基因多态性与动脉粥样硬化性疾病或卒中的关系成为近年来的研究热点。
The relationship between lipoprotein lipase (LPL) gene polymorphism and atherosclerotic diseases or stroke has become a topic of general interest in recent years.
大多数研究表明急性运动致骨骼肌脂蛋白脂酶(LPL)活性增强，脂肪组织LPL活性下降。
Recent studies show that LPL activity is increased in skeletal muscle at the same time it is reduced in adipose tissue after acute exercise.
腹型肥胖的发生与遗传、年龄、能量摄入、脂蛋白脂酶（LPL）活性及脂解活性等多种因素相关；
From the research in the abdominal obesity, we know it can be caused by many factors, such as the gene, age, energy intake, lipoprotein lipase (LPL) reactive and the lipolysis reactive.
抑制脂肪代谢相关酶脂蛋白脂酶、硬脂酰辅酶A 脱氢酶、脂肪酸合成酶等的活性来产生降脂作用。
CLA may inhibit the enzymes, lipoprotein lipase(LPL), stearoyl-CoA desaturase(SCD), fatty acid synthase(FAS)and so on, which are related to lipid metabolism.
烟酰水杨酸对鹌鹑实验性脂肪肝有明显的预防作用，这一作用与激活脂蛋白脂酶、抑制肝脂酶有密切关系。
The conclusion is that NSA has significant role of prevention function to experimental fatty liver model which is closely related to activated LPL and inhabit HL.
目的探讨人肾小球系膜细胞脂蛋白脂酶（LPL）的表达情况以及极低密度脂蛋白（VLDL）对其表达的影响。
Objective To study the expression of lipoprotein lipase(LPL) in human glomerular mesangial cells and the effect of very low-density lipoprotein(VLDL) on the expression of LPL.
结果糖尿病大鼠心肌脂蛋白脂酶mRNA是升高的，经BMOV治疗后心肌细胞脂蛋白脂酶m RNA表达可降低，基本恢复到正常对照水平。
Results The levels of LPL mRNA in cardiac myocytes of diabetic group increased, but though BMOV treatment the levels of LPL mRNA decreased to control levels.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
结论HP感染可引起脂代谢紊乱，可能机制为HP感染抑制了脂蛋白酶的活性与分泌。
Conclusion HP infection might cause disorder in the lipid metabolism. The possible mechanism might be that the HP infection can suppress activity and secretion of lipoprotein lipase.
结论：泰脂安对高脂模型大鼠有较好的降脂作用，其降脂作用机制可能与泰脂安升高血清中的脂蛋白脂肪酶含量有关。
CONCLUSION: Taizhi "an capsule can decrease the hyperlipidemia. Its mechanism is related to the regulation of lipid metabolism and improvement of the concentration of LPL (lipoprotein lipase)."
结论：泰脂安对高脂模型大鼠有较好的降脂作用，其降脂作用机制可能与泰脂安升高血清中的脂蛋白脂肪酶含量有关。
CONCLUSION: Taizhi "an capsule can decrease the hyperlipidemia. Its mechanism is related to the regulation of lipid metabolism and improvement of the concentration of LPL (lipoprotein lipase)."
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