它提供了300多个美国海岸受缺氧影响的水体综合清单。在八个案例研究中，突出介绍了一系列受缺氧影响有代表性的生态系统。
It provides a comprehensive list of the more than 300 U.S. coastal water bodies affected by hypoxia and, in eight case studies, highlights a range of representative ecosystems affected by hypoxia.
目的研究急性缺氧和慢性适应性缺氧对大鼠心脏收缩和舒张功能的影响。
Objective to study the effects of acute hypoxia and intermittent hypoxic acclimatization on cardiac systolic and diastolic functions in rats.
目的：通过测定化学性缺氧小鼠的存活时间，观察葡萄籽原花青素对化学性缺氧小鼠的影响。
AIM: To observe the effect of grape seed proanthocyanidins on the mice with chemical hypoxia by measuring the survival time of mice.
目的观察急性持续性高原缺氧条件下视网膜结构、功能变化，探讨牛磺酸对视网膜缺氧损伤的保护作用。
Objective To investigate retina structure and function under the condition of acute continuous hypoxia at high altitude and study on the protective effects of taurine on retina injury.
结果：益寿调脂片可使小鼠在常压缺氧或亚硝酸钠所致化学性缺氧条件下的存活时间和游泳存活时间显著延长，可明显提高小鼠的耐寒和耐高温能力；
Results Yishou Tiaozhi tablet could significantly postpone living times while mice was in normal or chemical hypoxia and swimming, and elevate tolerating cold and high temperature function in mice.
结论：主要的高危因素在产前及产时，缺氧性因素是主因，特别是宫内的慢性缺氧，是影响小儿智能发育的主要因素。
Conclusion: Main high risk factors lie in antepartum and intrapartum. It is anoxia that major cause to affect child's intelligence development, especially chronic hypoxia in uterus.
目的探讨川芎嗪对常压缺氧和化学性缺氧小鼠的保护作用。
Objective To research the protection action of ligustrazine for the normal pressure hypoxia and chemical hypoxia mice.
目的：探讨反复缺氧预激对小鼠缺氧耐受性的影响及其可能机制。
Objective:This study was to explore effects of repetitive hypoxic preconditioning on the myocardial tolerance to hypoxia of mice and its possible mechanism.
结果提示：高原移居平原者与世居平原者相比，脑血管缺氧性扩张反应较强，而肺血管缺氧性收缩反应较弱。
This study suggests that long term residing at high altitude results in an increase in cerebral vasodilative response and a decrease in pulmonary vasoconstrictive response to acute hypoxia.
目的研究吸入一氧化碳(CO)对急性缺氧性肺动脉高压及缺氧性肺损伤的影响。
Objective To investigate the effect of carbon monoxide (CO) inhalation on acute hypoxic pulmonary arterial hypertension and acute hypoxic lung injury.
结论音频电预防性治疗可改善由低压缺氧造成的记忆功能损害，并对急性缺氧所致的中枢抑制有改善作用。
Conclusion Preventive therapy of MET can improve learning and memory impairment and also improve central inhibition induced by hypobaric hypoxia.
本实验将大鼠随机分为4组：空白对照组、磁作用对照组、缺氧性心肌损伤组、缺氧性心肌损伤磁场治疗组。
In this experiment, the rats were randomly divided into 4 groups: blank control group, magnetic effect group (MK group), ischemic myocardial injury group (IMI group).
方法制备心肌组织缺氧损伤模型，观察不同剂量的地高辛抗血清对缺氧损伤心肌组织内源性洋地黄样因子水平和心肌细胞膜atp酶活性的影响。
Methods it was observed that different concentration anti_digoxin antiserum effect on endogenous digitalis_like factor and cell membrane ATPase activity in hypoxic myocardium model.
该文主要阐述细胞凋亡与新生儿缺氧缺血性脑损伤的关系，提出细胞凋亡机制在缺氧缺血性脑损伤中的重要性。
In this review, we summarized relationship between apoptosis and neonatal hypoxic-ischemic brain damage (HIBD) and importance of apoptosis mechanism in HIBD.
可采用低张性缺氧方法使孕鼠缺氧来制造胚胎大鼠宫内缺氧模型。
The model of hypoxia in the inner of the uterus could be established by making the conceiring female rat in hypotonic hypoxia.
目的探讨慢性缺氧引起缺氧性肺血管收缩反应降低的机制。
Objective To investigate the mechanism of chronic hypoxia induced decreased HPV.
结论急性低压缺氧可引起老龄大鼠大脑皮质和丘脑中et、NO含量明显升高以及大脑皮质血管内皮细胞和神经细胞明显损伤，ET、NO可能参与了缺氧性脑损害的病理过程。
Conclusion It is suggested that, the increase of ET and NO contents of cerebral cortex and thalamus may participate in the pathogenesis of hypoxia brain injury in old rats.
缺氧诱导因子- 1 (HIF - 1)是由低氧等诱导细胞产生的1种转录因子，能激活许多缺氧反应性基因的表达。
Hypoxia inducible factor-l (HIF-1), which can stimulate expression of hypoxia induced-responsed, is a kind of transcriptive factor.
本文主要介绍高级中枢、缺氧后脑血流量变化、缺氧对呼吸中枢神经元的实质性损害、神经递质和神经调质在缺氧性呼吸抑制过程中的作用。
This article introduces the effect of high central nervous system, changes of cerebral blood flow after hypoxia, damage of hypoxia to neurons in the respiratory center, neurotransmitters a…
有必要建立一种相关缺氧模型，用于HIF和缺氧耐受性的研究。
It is necessary to establish a hypoxia model for HIF and to perform further hypoxia tolerance research.
提示松寿丹不仅对单纯血液性缺氧有保护作用，而且还通过其显著的降血脂作用改善循环性缺氧，提高机体耐缺氧能力。
All the results imply that SSD not only has the protective effects on the acute hypoxia, but also significantly improve the circulated hypoxia by lowering the level of 1 IP id in the serum.
结论大鼠慢性缺氧时一氧化氮释放减少，而一氧化氮释放减少可能参与缺氧性肺动脉高压的形成。
Conclusion Nitric oxide release decreased during chronic hypoxia, which was probably involved in the mechanism of hypoxic pulmonary hypertension.
作者研究了慢性间断低压缺氧对大鼠肺血管反应性的影响及其与组胺的关系。动物经慢性缺氧后发生慢性肺动脉高压，其肺血管对急性缺氧的反应性降低。
The effect, of chronic intermittent hypobaric hypoxia on pulmonary vascular reactivity to acute hypoxia in. rats and its relationship to histamine have been studied.
作者研究了慢性间断低压缺氧对大鼠肺血管反应性的影响及其与组胺的关系。动物经慢性缺氧后发生慢性肺动脉高压，其肺血管对急性缺氧的反应性降低。
The effect, of chronic intermittent hypobaric hypoxia on pulmonary vascular reactivity to acute hypoxia in. rats and its relationship to histamine have been studied.
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