目的：运用DNA家族改组方法提高水蛭素的抗凝血酶活性。
AIM: To directed promote the antithrombotic activity of hirudin in vitro with DNA family shuffling method.
目的观察患者回到ICU后的抗凝血酶活性以及术后各种不良结果的联系。
AIM To investigate the association between antithrombin activity at admission to the ICU(ICU-antithrombin activity) and various outcome variables.
研究人员将抗凝血酶III上的肝素结合肽段黏附在称为聚乙二醇的人工合成材料上。
The researchers attached heparin-binding peptides from antithrombin III to a synthetic material called polyethylene glycol.
肝脏移植恢复期抗凝血酶活性和纤溶功能的恢复是衡量肝脏移植成功的较好指标之一。
We conclude that it is very important to measure AT:A, PLG:A and other coagulation parameters during the courses of liver transplantation.
结论静脉血栓形成可导致抗凝血酶活性下降，狼疮样抗凝物质增多是导致静脉血栓形成的因素之一。
Conclusions venous thrombosis can result in decreased antithrombin activity, and increased lupus_like anticoagulants is one of the factors that lead to venous thrombosis.
研究者利用了肝素（一种多糖）和血液中的抗凝血酶III（一种多肽片断）之间的相互作用来控制凝固时间。
Researchers have used the interaction between a polysaccharide called heparin and a peptide fragment of a protein called antithrombin III, which is contained in the bloodstream to control clotting.
杂合子i型(定量)和大多数II型(定性)抗凝血酶缺乏症使静脉血栓栓塞(VTE)的风险显着增加，而纯合型突变会导致死亡。
Heterozygous type I (quantitative) and most type II (qualitative) antithrombin deficiencies highly increase the risk of venous thromboembolism (VTE), while homozygous mutations are lethal.
杂合子i型(定量)和大多数II型(定性)抗凝血酶缺乏症使静脉血栓栓塞(VTE)的风险显着增加，而纯合型突变会导致死亡。
Heterozygous type I (quantitative) and most type II (qualitative) antithrombin deficiencies highly increase the risk of venous thromboembolism (VTE), while homozygous mutations are lethal.
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