结论：提示慢性氟中毒对大鼠学习记忆能力有显著的影响。
Conclusions: Fluorosis had significant damages on the rats' capacity of learning-memory.
结果表明，成功地复制出了不同程度的鸡慢性氟中毒模型；
The results showed as follows:The animal patterns of chick fluorosis were successfully established;
可以将血清羟脯氨酸含量的变化作为鸡慢性氟中毒早期诊断的主要参考指标。
So, The variation of serum Hyp content can be considered as an important and early index for diagnosing chick fluorosis.
结论：慢性氟中毒对大鼠心肌胶原代谢无明显影响，心肌胶原可能不是氟化物作用的靶子。
Conclusion Chronic fluorosis has no obvious effect on myocardial collagen metabolism of rats and myocardial collagen is not likely the main target of fluoride.
实验结果显示，慢性氟中毒大鼠脑组织中胆固醇和多萜醇含量与正常对照组相比，未见异常改变。
Results showed that no significant difference of cholesterol and dolichol contents in Brian tissues between rats with fluorosis and normal controls were detected.
目的观察慢性氟中毒对小鼠切牙成釉细胞骨形成蛋白- 2 (BMP - 2)表达的影响，探讨氟斑牙的致病机制。
Objective To investigate the expression of BMP-2 in mice incisor enamel organ cells under chronic fluoride exposure and elucidate the mechanism of dental fluorosis.
结果慢性氟中毒大鼠中缝大核5 -HT神经元减少，胞质内5 -HT阳性反应颗粒减少且界限不清，显微图像分析仪定量检测神经元内5 -HT含量降低。
Results The results showed that the number of 5-HT positive neurons in nucleus raphe magnus decreased in fluorosis group with the declared 5-HT positive particles in cy…
慢性氟暴露会导致由于低水平的系统吸收引起的在骨骼结构里的异常钙积累(氟中毒)。
Chronic fluorine exposure may cause abnormal calcium accumulation in bone structure (fluorosis) due to low-level systemic absorption.
慢性氟暴露会导致由于低水平的系统吸收引起的在骨骼结构里的异常钙积累(氟中毒)。
Chronic fluorine exposure may cause abnormal calcium accumulation in bone structure (fluorosis) due to low-level systemic absorption.
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