• 氯丙嗪心肌缺血性损伤保护作用机制与抑制PLA_2活性自由基的作用有关。

    The results suggest that chlorpromazine had a protective effect on membrane damage of ischemic myocardium, which probably resulted from its inhibiting PLA_2 activity and resisting free radicals.

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  • 已经确证心肌收缩始于动作电位使肌去极化。

    It is well established that the cardial contraction is initiated when an action potential depolarizes the sarcolemma.

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  • 已经确证心肌收缩始于动作电位使肌去极化。

    It is well established that the cardiac contraction is initiated when an action potential depolarizes the sarcolemma.

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  • 分离心肌线粒体测定流动性(lfu)。

    Rat myocardial mitochondria were isolated, and membrane lipid fluidity (LFU) was determined.

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  • 我们的研究证实胆固醇可以调节离子通道使之随时补充心肌细胞表面结构。

    Here we show that cholesterol regulates the submembrane pool of ion channels readily available for recruitment into the surface membranes of heart cells.

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  • 心律失常发生机制药物抗心律失常作用机制研究,也聚焦于对心肌细胞离子通道及跨离子流的研究。

    The studies of the mechanisms of arrhythmogenesis and the action of antiarrhythmic drugs have already focused on the events of ionic channel currents in isolated single heart cells.

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  • 目的探讨风湿性心脏病心肌细胞线粒体磷脂损伤术后血量关系

    Objective To explore the relationship between myocytic mitochondrial membrane phospholipid(mtMPL) injure of rheumatic heart disease and low cardiac output following cardiac valve replacement.

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  • 循环儿茶酚升高心肌细胞产生直接毒性作 用、增加通透性心肌纤维化,这些作用可引起细胞死亡。

    The increase of circulating catecholamines exerts direct toxic effect on cardiac myocytes, increases membrane permeability and myocardial fibrosis;

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  • 敬钊毒素-I(JZTX-I)一种能够抑制心肌通道活的新型蜘蛛神经毒素,该文结合高效液相色谱色氨酸荧光测定技术研究了JZTX-I的磷脂结合活性。

    Jingzhaotoxin-I(JZTX-I), a 33-residue polypeptide with three disulfide bonds, was a novel spider neurotoxin preferentially inhibiting cardiac sodium channel inactivation.

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  • 方法应用高频超声检测19扩张型心肌病患者18正常对照者动脉弹性和内中复合体厚度(IMT)。

    Methods Carotid arterial compliance and IMT were measured by high-frequency ultrasonography in 19 patients with dilated cardiomyopathy and 18 healthy subjects.

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  • 结果术后10手术组心肌粘着、桥间隙连接结构模糊,连接间隙增宽;

    Results At the 10th week after constriction, the structure of cadherin, desmosome and gap junction in intercalated discs were destroyed, gap of intercellular space widened.

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  • 应用玻璃电极记录心肌细胞动作电位.研究脂布福吉宁(RBG豚鼠心室的电药理学作用。

    Electropharmacological effects of resibufogenin(RBG)on guinea pig papillary muscle were studied by means of conventional glass microelectrode technique.

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  • 应用玻璃电极记录心肌细胞动作电位.研究脂布福吉宁(RBG豚鼠心室的电药理学作用。

    Electropharmacological effects of resibufogenin(RBG)on guinea pig papillary muscle were studied by means of conventional glass microelectrode technique.

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