研究者们利用小鼠晶胚来培育脑组织，并且设定了一种环境，在此环境下，脑组织会逐渐流失左旋多巴胺神经细胞，这与帕金逊症的症状非常相似。
Using tissue from mouse embryos, researchers prepared brain cultures using conditions that favor the slowly progressing loss of dopamine neurons, a hallmark of the disease.
经常患者拿着左旋多巴的处方，左旋多巴在脑细胞中在酶的作用可以转变为多巴胺。
Often patients are given prescriptions for levodopa, or L-dopa, which is converted into dopamine by enzymes in the brain.
小剂量左旋多巴和多巴胺激动剂治疗有效。
Low dose levodopa and dopamine agonists are effective in controlling most symptoms of the disorder.
结论：大剂量左旋多巴干预能明显改善PD大鼠的旋转行为，并使纹状体多巴胺d2受体明显下调。
CONCLUSION: After high dose of L-dopa intervention, rotation behavior of PD rats improves, and D2 receptor is down-regulated significantly.
目前，帕金森氏症患者多服用名为左旋多巴的药物来重新调整大脑内多巴胺的浓度，但成功率因个体差异有高有低。
At present, most patients take a drug called L-dopa to readjust levels, but with varying levels of success.
目前，帕金森氏症患者多服用名为左旋多巴的药物来重新调整大脑内多巴胺的浓度，但成功率因个体差异有高有低。
At present, most patients take a drug called L-dopa to readjust levels, but with varying levels of success.
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