The mechanism of myocardium ischemia reperfusion injury involve in generating of reactive oxygen species, overload of Ca2 + and releasing of catecholamine by ischemia.
心肌缺血再灌注损伤的主要机制为氧自由基大量释放，细胞内钙超载以及缺血导致的大量儿茶酚胺释放。
Objective To observe the protective effect of ischemic preadaptation on myocardium injury of ischemia reperfusion by adopting the animal model of ischemia reperfusion on myocardium injury rabbits.
目的采用家兔心肌缺血再灌注损伤动物模型，观察缺血预适应对心肌缺血再灌注损伤的心肌保护作用。
Objective: to explore research the protective effect of ischemia preconditioning to rabbits 'myocardium injury of ischemia reperfusion, and discuss its mechanism.
目的：从细胞保护角度出发，研究缺血预适应对家兔心肌缺血再灌注损伤的保护作用，并探讨其机制。
AIM: To study whether magnesium chloride plays a protective role in myocardium of rats with ischemia reperfusion injury, and investigate the cardioprotective action mechanism of magnesium chloride.
目的：研究氯化镁对大鼠缺血再灌注损伤心肌是否具有保护性作用，同时探讨氯化镁的心肌保护作用机制。
Results (1)Level of myocardium enzyme with ischemia reperfusion group is significantly different compared to ischemia preadaptation(P<0.01).
结果（1）缺血预适应组心肌酶水平与缺血再灌注组相比差异均极为显著（P<0.01）；
Conclusion: Valerian extract can protect myocardium from ischemia reperfusion injury.
结论：缬草提取物有抗心肌缺血再灌注损伤的作用。
Aim To investigate the protective effect of fluvastatin on ischemia reperfusion myocardium in normocholesterolemic rabbit.
目的观察氟伐他汀对正常血脂兔的心肌缺血再灌注损伤有无保护作用及其可能原因。
Conclusion IL-8 is released during reperfusion of myocardial ischemia. It might induce infiltration and injury of neutrophils for myocardium. Its clinical significance should be further investigated.
结论IL - 8在心肌缺血和再灌注期间释放，并诱导了中性粒细胞致心肌的浸润与损伤，其在临床的意义有待进一步探讨。
Conclusion Beating heart on cardiopulmonary bypass with mild hypothermia could attenuate ischemia-reperfusion injury of myocardium, so it has better myocardial protection effects.
结论浅低温心肺转流心脏不停跳心内直视手术可减轻心肌缺血-再灌注损伤，有较好的心肌保护作用。
Remote organ ischemic preconditioning (RPC) may reduce heart infarct size following myocardium ischemia and reperfusion.
远端器官缺血性预处理(RPC)可减少缺血再灌注后心肌坏死范围。
A model of acute ischemia and reperfusion of the myocardium was developed in cats in vivo.
建立猫在体心脏急性心肌缺血再灌注模型。
Aim to investigate the protective effects of MLT (melatonin) on acute ischemia-reperfusion induced myocardium damage in rats in vivo.
目的探讨外源性褪黑素(MLT)对在体大鼠心肌急性缺血再灌注损伤的保护作用。
Objective to observe whether the heat-shock pretreatment could restrain the expression of apoptosis cells in ischemia-reperfusion myocardium of rats.
目的观察热休克预处理对大鼠缺血再灌注损伤心肌细胞凋亡表达的影响。
Objective To investigate the protective effects of Puerarin and glucose injection (PGI) on the myocardium during myocardial ischemia and reperfusion injury (MIRI) and its mechanism.
目的探讨葛根素葡萄糖注射液(PGI)对缺血-再灌注损伤兔心肌的保护作用及其机制。
This study may be indicative of the mechanism about ischemia-reperfusion injury to diabetic myocardium and the therapy for diabetic patients with ischemic heart disease.
本研究结果可能有助于提示糖尿病条件下的缺血-再灌注心肌损伤机制以及对合并缺血性心脏病的糖尿病患者的治疗方案。
AIM: To investigate the changes in nuclear calcium content and permeability of nuclear pore complex in rat myocardium during ischemia reperfusion injury.
目的：探讨心肌缺血再灌注损伤时心肌细胞核钙含量与核孔复合体通透性的变化及其相互关系。
Objective To study the protective effects and mechanism of Egb on endothelial cell injury induced by myocardium experienced ischemia-reperfusion in rabbits.
目的研究银杏达莫对兔心肌缺血再灌注损伤内皮细胞的保护作用。
Objective To study the effects and mechanism of Huangqi against myocardium injury induced by ischemia and reperfusion in rats.
目的探讨黄芪对大鼠缺血再灌注心肌的保护作用及其机制。
Methods A rabbit model of myocardial ischemia and reperfusion injury was established, and HSP70mRNA in myocardium was detected by in situ hybridization histochemistry.
方法建立家兔心肌缺血再灌流损伤模型，通过原位杂交技术观察研究。
Objective To investigate the effects of renal ischemic postconditioning(Renal PostC) on myocardium enzymes in rabbits with acute myocardial ischemia and reperfusion.
目的：观察白附片对正常大鼠和肾阳虚证大鼠心肌酶的影响，探索其对两种大鼠心脏毒性的差异。
Objective To investigate the effects of renal ischemic postconditioning(Renal PostC) on myocardium enzymes in rabbits with acute myocardial ischemia and reperfusion.
目的：观察白附片对正常大鼠和肾阳虚证大鼠心肌酶的影响，探索其对两种大鼠心脏毒性的差异。
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