Conclusion In the process of liver transplantation, the ischemia reperfusion damage may lead to hepatic microcirculation disturbance, which is a major cause of graft failure.
结论肝移植过程中，缺血再灌注损伤可造成肝脏微循环紊乱，是供肝失活的一个重要原因。
It has broad pharmacological functions such as to prevent oxygenated stress damage, to diminish inflammation, to drop high blood pressure and to resist ischemia reperfusion injury.
此外它还具有广泛的药理学作用，例如防止氧化应激损伤，减轻炎症反应，降低血压，抗缺血再灌注损伤等。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
CONCLUSION: Paeonol may inhibit ICAM 1 protein expression after focal cerebral ischemia reperfusion in rats so as to relieve neuron damage.
结论：丹皮酚可能具有抑制大鼠脑缺血再灌注后ICAM -1蛋白表达的作用，从而减轻了神经元损伤。
Objective:To study the preventive effect of damage of ischemia reperfusion in rat liver by applying Shenmai Injection.
目的：研究参麦注射液对肝缺血再灌注损伤的保护作用。
The myocardial ischemia and ischemia reperfusion all damage myocardial collagen fibers.
缺血、缺血再灌注均使心肌胶原网络破坏。
The said kidney disease in this invention includes of chronic kidney disease, diabetic nephropathy, kidney damage coursed by acute ischemia reperfusion.
本发明所述的肾脏疾病包括慢性肾脏疾病、糖尿病肾病、急性缺血再灌注导致的肾脏损伤。
Aim: Damage of endothelial cells and adhesion of leukocytes and platelets were studied in rat mesentery microvessels after ischemia reperfusion.
目的：在活体上探讨缺血再灌后血管内皮细胞损伤及白细胞、血小板与内皮之间粘附的变化。
OBJECTIVE To investigate the protective mechanism of adenosine in renal damage of ischemia reperfusion.
探讨腺苷对肾脏缺血再灌注损伤的保护作用机制。
Recent studies have demonstrated that EPO has protective effects on myocardial infarction, ischemia-reperfusion and adriamycin-induced myocardial damage.
最近的研究提示，EPO对心肌梗死、缺血再灌注损伤及阿霉素心肌损害等具有保护作用。
Objective to investigate the relationship between the apoptosis of intestinal epithelium and characteristics of intestinal mucosal ischemia-reperfusion damage and repair after injury in rats.
目的探讨小肠黏膜缺血损伤及再生修复的特征及规律，以及与肠上皮细胞凋亡的关系。
Conclusion BQ-123 is useful in preventing the neuronal damage following the cerebral ischemia reperfusion in rats.
结论内皮素受休拮抗剂BQ- 123对全脑缺血再灌流引起海马区神经元损伤有部分保护作用。
Lung ischemia-reperfusion injury inevitably occurs within the first 72 hours after lung transplantation and is typically characterized by nonspecific alveolar damage, lung edema, and hypoxemia.
在肺移植中不可避免地要发生肺缺血-再灌注损伤，这种损伤以肺移植后第一个72小时内发生的非特异性肺泡损伤、肺水肿和低氧血症为特征。
CONCLUSION Adenosine could protect the damage of ischemia reperfusion.
腺苷对大鼠肾脏缺血再灌注损伤有保护作用。
Objective To investigate the transcription-coupled repair factor CSB/ERCC6 expression and neuronal DNA damage in ischemic area after cerebral ischemia-reperfusion in rats.
目的探讨大鼠脑缺血再灌注后缺血区不同时相转录修复偶联因子（CSB/ERCC6 ）表达对神经元DNA损伤的作用。
Ischemic tolerance refers to the adaptive response to transient ischemia and reperfusion, which can improve tissue tolerance during the following damage caused by more severe ischemic events.
缺血耐受是指对短暂性缺血和再灌注的适应性反应，提高组织对随后较长时间缺血和再灌注的耐受力。
These results suggest that oxygen free radicals may damage the activity of cytochrome oxidase, the reduction of which may play an important role in the injury of renal ischemia and reperfusion.
提示氧自由基可能损害细胞色素氧化酶活性，细胞色素氧化酶活性降低在肾缺血再灌流损伤中可能起重要作用。
Aim to investigate the protective effects of MLT (melatonin) on acute ischemia-reperfusion induced myocardium damage in rats in vivo.
目的探讨外源性褪黑素(MLT)对在体大鼠心肌急性缺血再灌注损伤的保护作用。
Objective The effects of YVAD-FMK on neuron damage after complete cerebral ischemia-reperfusion injury in rats were studied.
目的研究YVAD - FMK对大鼠全脑缺血再灌注后神经元的影响。
Objective To explore the survival mechanism of hippocampal neurons after the damage of hypoxic ischemia and reperfusion of the brain.
目的探讨缺氧缺血再灌注后海马神经元的存活机制。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Conclusion: Damage of epiphysis could be aggravated after ischemia and reperfusion injury.
结论：缺血再灌注可加重幼年兔股骨头骨骺骨细胞损伤。
Hyperglycemia significantly increases brain damage, mortality rate, and long-term disability after cerebral ischemia and reperfusion injury.
高血糖症会显著增加脑损伤，导致病死率升高，并由脑缺血再灌注损伤后导致患者长期残疾。
Hyperglycemia significantly increases brain damage, mortality rate, and long-term disability after cerebral ischemia and reperfusion injury.
高血糖症会显著增加脑损伤，导致病死率升高，并由脑缺血再灌注损伤后导致患者长期残疾。
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