Adenosine stimulated a significant arteriolar nitric oxide (NO) production and dilation, which were sensitive to NO synthase inhibitor L-NMMA.
腺苷能够显著激发动脉一氧化氮(NO)的产生而使血管舒张,且对一氧化氮合酶抑制剂L -NMMA 十分敏感。
Adenosine stimulated a significant arteriolar nitric oxide (NO) production and dilation, which were sensitive to NO synthase inhibitor L-NMMA.
腺苷能够显著激发动脉一氧化氮(NO)的产生而使血管舒张,且对一氧化氮合酶抑制剂L -NMMA 十分敏感。
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