The relationship of codon 54 variation in intestinal fatty acid binding protein gene (FABP2) with NIDDM and its pathogenesis, and also with obesity, was investigated in 231 Chinese.
在231例中国人中观察小肠脂酸结合蛋白基因(FABP2)54号密码子变异与NIDDM及其病理生理以及肥胖的关系。
Results The distribution of IRS1 gene polymorphism showed no difference between patients with NIDDM and normal controls.
结果IRS1基因多态性在NIDDM和正常人中的分布没有明显差异。
NIDDM were collected. Exon 17 ~ 21 of insulin receptor gene were amplified by PCR and screened by SSCP electrophoresis. After silver staining, the mutation samples were sequencing.
选择89例非胰岛素依赖性糖尿病(NIDDM)患者，扩增胰岛素受体基因的外显子17 -21,sscp电泳，银染色后，对突变样品进行序列分析。
NIDDM were collected. Exon 17 ~ 21 of insulin receptor gene were amplified by PCR and screened by SSCP electrophoresis. After silver staining, the mutation samples were sequencing.
选择89例非胰岛素依赖性糖尿病(NIDDM)患者，扩增胰岛素受体基因的外显子17 -21,sscp电泳，银染色后，对突变样品进行序列分析。
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