It was also found that the interaction of the exuberance of internal heat toxin and fu-organ turbidness stagnation enlarged the risk of the onset of dementia after basal ganglia infarction.
且热毒内盛和腑滞浊留两者相互作用，提高了基底节梗塞后痴呆发生的危险性。
It was also found that the interaction of the exuberance of internal heat toxin and fu-organ turbidness stagnation enlarged the risk of the onset of dementia after basal ganglia infarction.
且热毒内盛和腑滞浊留两者相互作用，提高了基底节梗塞后痴呆发生的危险性。
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