The brain tissue changes in mouse model with brain hemorrhage, included dropsy in different degree, inflammatory cell infiltrate, nerve cell necrosis and vascular proliferation as well as gliacyte.
大鼠模型脑出血后脑组织的改变包括血肿周围存在不同程度的水肿、炎性细胞浸润、神经元坏死及胶质细胞和血管增生。
The mechanism of improved tissue viability is unclear, but the anti-inflammatory effects may alter the relative contribution of tissue necrosis versus apoptosis to cellular injury.
机制尚不清楚，但其抗炎作用可能改变了组织坏死和细胞凋亡在细胞损伤中所占的比例。
In sodium bicarbonate group, large amounts of inflammatory ooze was found to adhere to the surface, with necrosis of hair follicles, a few lymph cells infiltrated and hyperemia oedema disappeared.
碳酸氢钠组表面大量炎性渗出物附着，伴有毛囊坏死，少许淋巴细胞浸润，充血水肿消失；
ECG records the changes in light microscopy myocardial necrosis and infiltration of inflammatory situation, and the separation of serum testing, such as myocardial enzyme.
记录心电图的改变，在光镜下观察心肌细胞的炎性浸润和坏死情况，用分离的血清检测心肌酶等。
Objective to investigate the role of tumor necrosis factor-a (TNF-a), norepinephrine (ne), endothelin (ET), adrenomedullin (ADM) in the pathogenesis of systemic inflammatory response syndrome (SIRS).
目的探讨肿瘤坏死因子- a (TNF - a)、去甲肾上腺素(NE)、内皮素(et)、肾上腺髓质素(adm)在全身炎症反应综合征(SIRS)病理过程中的作用。
The alveolar edema and lung hemorrhage were the severest in the HTP group. The lung necrosis and the inflammatory cells infiltration were the severest in the HTP and HTNP group.
病变中肺泡水肿、肺出血及肺充血以HTP组最严重，肺组织坏死及炎性细胞浸润以HTNP及HTP组最明显。
Results The main pathological features of IC are hyperemia, edema, hemorrhage, degeneration and necrosis of bowel wall with various degrees of inflammatory reaction.
结果缺血性肠炎的病理改变以肠壁充血、水肿、出血及变性坏死为主，伴不同程度的炎症反应。
In muscle biopsies there are inflammatory cells infiltration, muscle fiber necrosis accompanying regeneration and connective tissue proliferation.
肌组织可见炎性细胞浸润、肌纤维变性、坏死与再生及结缔组织增生。
The post-operative pathological examination showed varying degrees of apomorphosis and necrosis of tumor cell, intercellular substance edema, fibrous hyperplasia and inflammatory cell invasion.
术后病理学检查发现，观察组癌细胞均有不同程度的变性、坏死，细胞间质水肿，纤维增生，炎性细胞浸润；
After treated by acupuncture, the necrosis areas reduced and the degree of the adhesiveness and infiltration of the inflammatory cells also decreased.
而应用针刺治疗后各组缺血坏死灶范围减小，半影区炎性细胞粘附及浸润的程度明显减轻。
The histologic changes of renal tissue were interstitial edema with inflammatory cellular infiltration, denaturalization and necrosis in some tubular epithelia. LPS caused 20% rats death.
肾间质水肿伴炎症细胞浸润，部分小管上皮细胞变性、坏死并见管型，死亡率2 0 %。
AA was also found by pathological observation, with swelling, necrosis in the surface synovial tissues. Edema and diffusive infiltration of inflammatory cells happening in connective tissue as well.
模型组关节液白细胞计数显著高于对照组，病理学观察有急性炎症发生，表现为表面滑膜组织肿胀、坏死、结缔组织水肿、炎症细胞弥漫性浸润等；
The intramuscular implantation in mice induced no muscular necrosis or fibrous capsule formation, except for slight inflammatory reaction.
小鼠肌内埋置后植入部位无肌肉坏死，炎症反应轻，无纤维包裹。
The inflammatory cells and necrosis of hepatic cells were significantly reduced compared with those in caudal vein group.
经肝动脉移植组及阳性对照组病理学上改善最明显，肝内炎症细胞浸润及肝细胞坏死均明显减少。
The causes were penetrating trauma in 22, blunt trauma in 2, iatrogenic injury in 2, atherosclerosis in 6, medical necrosis in 6, and inflammatory in 2 cases.
内锐器伤所致22例，钝性伤2例，医源性损伤2例，感染2例，动脉粥样硬化和动脉中层坏死所引起的各6例。
The myocardial of R group with inflammatory cell infiltration and the extent of necrosis was lighter than M group, but seriouser than S group.
利巴韦林组心肌炎性细胞浸润和坏死程度比模型组轻，比中药治疗组重。
Conclusions: in winter, tumor necrosis factor antagonists treatment does not increase the risk of upper respiratory tract infection in inflammatory arthritis patients.
结论：肿瘤坏死因子拮抗剂的应用未使冬季上呼吸道感染的风险增加，其治疗炎性关节炎的安全性较好。
Acute Retinal Necrosis Syndrome (ARNS) is an inflammatory retinopathy which is rarely seen in clinic.
急性视网膜坏死综合征(ARNS)是一种严重影响视力的视网膜感染性疾病。临床少见。
Objective to investigate the effect of inflammatory cytokines interleukin-6 (IL-6), interleukin-8 (IL-8), interleukin-10 (IL-10) and tumor-necrosis factor (TNF) in multiple injury.
目的探讨血清炎性因子白介素- 6 (IL - 6)、白介素- 8 (IL - 8)、白介素- 10 (IL - 10)及肿瘤坏死因子(TNF)在多发伤中所起的作用。
Objective to investigate the effect of inflammatory cytokines interleukin-6 (IL-6), interleukin-8 (IL-8), interleukin-10 (IL-10) and tumor-necrosis factor (TNF) in multiple injury.
目的探讨血清炎性因子白介素- 6 (IL - 6)、白介素- 8 (IL - 8)、白介素- 10 (IL - 10)及肿瘤坏死因子(TNF)在多发伤中所起的作用。
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