The lithium-pilocarpine induced SE was used as the SE model.
建立氯化锂-匹罗卡品大鼠SE模型。
Conclusions PTZ -induced se can increase the dentate granule cell neurogenesis in the developing rat. NMDAR plays an important role in neurogenesis following seizures.
结论癫癎发作可增加幼鼠齿状回颗粒细胞的神经发生，而NMDAR在癫癎后的神经发生中起着促进作用。
RESULTS: Neuron necrosis, apoptosis, increase of apoptosis protein expression and proliferation of glial cells were the characteristics of pathological injury in the pilocarpine induced se models.
结果：毛果芸香碱惊厥持续状态模型鼠病理损伤特点为神经元坏死、细胞凋亡、凋亡蛋白表达增多，胶质细胞增生。
Objective: to observe the inhibitory effects of se on diethylnitrosamine (DEN) induced hepatocarcinogenesis.
目的：观察硒对二乙基亚硝胺(DEN)诱发大鼠肝癌的影响。
Objective:To observe the effect of selenium(Se), vitamin E and iodine on diet-induced obese mices.
目的：探讨硒、维生素E和碘对营养性肥胖小鼠的影响。
Objective To investigate the influence of selenium(Se) on oxidative damage, apoptosis and ultrastructure of human thyrocyte induced by H_2O_2 culture in vitro.
目的研究硒对H_2O_2诱导的体外培养人甲状腺细胞氧化损伤、凋亡及细胞超微结构的影响。
Objective: to study the protective effect in CCl4-induced liver injury by organoselenium from Se-enriched lactobacillus.
目的：研究乳酸菌源有机硒保护肝损伤组织脂质过氧化反应及其机制。
Objective: to study the protective effect in CCl4-induced liver injury by organoselenium from Se-enriched lactobacillus.
目的：研究乳酸菌源有机硒保护肝损伤组织脂质过氧化反应及其机制。
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