Objective to investigate the role of cytokine-induced neutrophil chemoattractant (CINC) in the pathogenesis of severe acute pancreatitis (SAP).
目的探讨中性粒细胞趋化因子(CINC)在重症急性胰腺炎(SAP)发病机制中的作用。
Conclusions: Exogenous HGF can attenuate cerulein-induced mild acute pancreatitis in mice. Its mechanism may be related to its effects on arachidonic acid metabolism.
结论：外源性HGF能减轻雨蛙肽诱导的小鼠轻症急性胰腺炎，其机制可能与对花生四烯酸代谢的影响有关。
The dynamic changes of the plasma and the tissue lipidpcroxidant(LPO) in the rats with experimental acute necro—hemorrhagic pancreatitis induced by bile salt and trypsin were observed.
本文观察了大鼠实验性急性胰腺炎时血浆和组织脂质过氧化物（LPO）的动态变化。
Methods: A model of experimental PCH induced acute hemorrhagic necrotizing pancreatitis(AHNP) was induced by retrograde injection of 5% sodium taurocholate into the pancreatic duct.
方法：应用5 %牛磺胆酸钠逆行胰胆管注射诱发大鼠AHNP并发脑组织损害模型。
Methods: Acute pancreatitis model was induced by intraperitoneal injection of cerulein or retrograde infusion of 5% sodium taurocholate into the bilipancreatic duct in SD rats.
方法： 分别采用腹腔注射雨蛙肽和胆胰管逆行注射5%牛磺胆酸钠建立大鼠轻症、重症急性胰腺炎模型。
Objective: to establish the model of fulminant acute pancreatitis (FAP) -associated liver injury induced by L-arginine in rats, and to study the mechanism of hepatic lesion in the model.
目的：建立L -精氨酸诱导大鼠暴发性胰腺炎(FAP)肝损伤模型，并探讨FAP肝损伤机制。
Objective: to establish the model of fulminant acute pancreatitis (FAP) -associated liver injury induced by L-arginine in rats, and to study the mechanism of hepatic lesion in the model.
目的：建立L -精氨酸诱导大鼠暴发性胰腺炎(FAP)肝损伤模型，并探讨FAP肝损伤机制。
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