Administration of the RXR agonist 9-cis-retinoic acid to demyelinated cerebellar slice cultures and to aged rats after demyelination caused an increase in remyelinated axons.
向脱髓鞘的小脑切片培养物和脱髓鞘后的衰老大鼠给予RXR激动剂9-顺式-视黄酸能够增加轴突髓鞘再生。
Administration of the RXR agonist 9-cis-retinoic acid to demyelinated cerebellar slice cultures and to aged rats after demyelination caused an increase in remyelinated axons.
向脱髓鞘的小脑切片培养物和脱髓鞘后的衰老大鼠给予RXR激动剂9-顺式-视黄酸能够增加轴突髓鞘再生。
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