When there is marked cellular injury, there is cell death.
细胞损伤严重时，可导致坏死发生。
This enzyme leaks from damaged cells and can indicate inflammatory or other cellular injury.
从损坏的细胞泄漏这种酶，可以表明炎症或其他细胞损伤。
The mechanism of improved tissue viability is unclear, but the anti-inflammatory effects may alter the relative contribution of tissue necrosis versus apoptosis to cellular injury.
机制尚不清楚，但其抗炎作用可能改变了组织坏死和细胞凋亡在细胞损伤中所占的比例。
Oxidant injury, or oxidative stress, occurs when highly reactive molecules called free radicals attack and damage cellular proteins, lipids (fats) and DNA.
氧化损伤，或氧化应激，出现在被称为自由基的高活性分子攻击和破坏细胞蛋白质，脂类(脂肪)和脱氧核糖核酸时。
Objective to establish a model of traumatic brain injury for studying hippocampal cellular apoptosis.
目的建立用于研究创伤性脑损伤诱导海马细胞凋亡的模型。
Conclusions Different molecular chaperones respond to immune injury according to their own characteristics after liver transplantation, which shows the mechanism of cellular self-protection.
结论不同种类的热休克蛋白依据自身表达的特点对移植后的免疫损伤作出反应，体现了细胞的自我保护机制。
Conclusion the cause of cellular immune reactivity was depressed after acute spinal cord injury was different from others of general injury.
结论急性脊髓损伤后细胞免疫反应受抑制的原因可能与一般的创伤不完全相同。
After brain injury by ischemia, the production of reactive oxygen species (ROS) may increase, leading to tissues damage via several different cellular molecular pathways.
缺血性脑损伤后，活性氧自由基(ROS)增加，以不同的细胞分子机制引起组织损伤。
Conclusion the cytoskeleton injury could cause the increase of cellular fragility and the decrease of the viscoelasticity, which ultimately lead to the change of cellular biodynamics.
结论细胞骨架的损伤可引起细胞脆性增加、粘弹性下降，导致细胞生物力学特性的改变。
Along with the increase of HSP, cellular survival rate obviously rose and heat injury was reduced.
随着HSP的增多，细胞存活率明显升高，受损伤程度减轻。
CF are an important cellular component of myocardial responses to injury and the source of paracrine growth factors.
CF是心肌损伤反应的重要细胞成分，并能产生旁分泌生长因子。
Cardiac remodeling is defined as an alteration in anatomy, inter diameter and function of cardiac Chambers caused by a series of molecular and cellular events after injury to the heart.
心脏重构是指发生于心肌损伤之后的分子和细胞事件所导致的心脏各腔室形态、内径和功能改变。
Meanwhile, the protein expression of HSP60 is reduced significantly, indicating that the decrease in the cellular protection of HSP60 may involve in the injury of pancreatic tissues.
同时，胰腺组织HSP60蛋白表达降低，提示HSP60细胞保护作用的减弱可能参与该损伤过程。
The pathogenesis of renal diseases has been implicated in the injury to immune system, tumorigenesis, inflammation and cellular toxic damage, which are the targets of many interventions.
肾脏疾病的发病机理涉及免疫﹑肿瘤﹑炎症﹑细胞毒损伤以及其他途径的损伤，并已成为各种干预治疗的目标。
Objective:To explore the mechanism and significance of myocardial cellular membrane damage following severe burn injury.
目的：探讨严重烧伤后心肌细胞膜损伤的机制及意义。
Conclusion SAB can protect cells from injury of glucose deprivation and the mechanism is related with its effect of mediating cellular calcium ion.
结论SAB对细胞缺糖损伤有明显的保护作用，其作用机制与抑制钙离子内流有关。
In the 1980s with the rapid development in neurobiology, research of peripheral nerve injury was deepened into cellular and molecular levels.
80年代以来，随着神经生物学的飞速发展，周围神经再生的基础研究又了长足的进步，已经进入了细胞、分子水平。
Hypoxic injury is an common and important pathological process. As a widely-studied molecule, P53 plays a significant role in variety of vital cellular reactions including hypoxia-induced injury.
缺氧损伤是常见而又重要的病理过程，P 53蛋白在缺氧调控中发挥的作用也逐渐为人们所认识。
Objective To study the change principles of the oxygen free radical on the sub-cellular level in the process of ischemia-reperfusion injury.
目的亚细胞水平上探索肝脏缺血再灌注损伤中氧自由基的变化规律。
Objective To study the change principles of the oxygen free radical on the sub-cellular level in the process of ischemia-reperfusion injury.
目的亚细胞水平上探索肝脏缺血再灌注损伤中氧自由基的变化规律。
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