The expressions of the various genes modulated cell growth and proliferation, apoptosis, cell cycle, stress response, transporters, chaperones, transcription regulation and drug metabolism.
差异表达的基因的功能涉及细胞生长增殖调控、凋亡、细胞周期、应激反应、转运及代谢酶等方面。
Conclusion FHIT gene may play a role in the regulation of cell proliferation of lung cancer, and the reduction in FHIT expression may be related to the pathogenesis of lung cancer.
结论FHIT基因可能参与肿瘤细胞增殖的调控，FHIT基因表达下降可能与肺癌的发生密切相关。
Objective: the relation, under the regulation of cell cycle, between cell apoptosis and proliferation during the onset and development of carcinoma was studied.
目的：探讨肝癌发生过程中细胞周期调控下细胞增殖与凋亡的关系。
These results led to the suggestion that the action of se involves in the regulation of cell differentiation and proliferation.
该结果表明硒可能涉及对细胞分化与增殖的调控。
PI3K becomes the focus on the effects of cell growth and cell proliferation by nutrition uptaking and cell cycle regulation.
PI3K调控细胞周期、参与营养物质的摄取，进而具有影响细胞的生长和分化等多方面功能，备受瞩目。
Conclusion: Cells proliferation and programmed cell death are interactive with each other in the regulation of the development of TMJ.
结论：细胞增殖与程序性细胞死亡密切相关，协同参与调控颞下颌关节的生长发育与塑形。
The expression of LRP16 is correlated with cell Proliferation of Prolactinomas and LRP16 may contributet to the regulation of cell Proliferating.
③LRP16基因的表达量与垂体泌乳素腺瘤细胞增殖活性存在相关关系，LRP16基因可能参与垂体泌乳素腺瘤细胞增殖的调控。
Activator Protein-1(AP-1) is a kind of trans-regulation factor, whose activation can participating in many biological effect including in cell cancerization, proliferation, apoptosis and so on.
激活蛋白-1（AP-1）是一种反式调节因子，它的活化参与细胞的癌变、增殖以及凋亡等多种生物学效应。
ConclusionATRA could inhibit proliferation of yolk sac tumor cell line, and down regulation of VEGF could be the inhibitory mechanism of ATRA on yolk sac tumor cell line in vitro.
结论全反式维甲酸可以抑制睾丸卵黄囊瘤细胞的增殖，抑制肿瘤增殖机制可能与下调VEGF的表达有关。
Conclusion Neoadjuvant chemotherapy with ET regimen is effective, and it may inhibit the proliferation of breast cancer cell by down-regulation of the expression of MCM7.
结论ET方案新辅助化疗有较好的疗效，可能通过抑制MCM 7蛋白的表达来阻止乳腺癌细胞的增殖。
Conclusion Neoadjuvant chemotherapy with ET regimen is effective, and it may inhibit the proliferation of breast cancer cell by down-regulation of the expression of MCM7.
结论ET方案新辅助化疗有较好的疗效，可能通过抑制MCM 7蛋白的表达来阻止乳腺癌细胞的增殖。
应用推荐