Methods: Fourvessel occlusion was used to establish the rat global brain ischemic model.
方法：利用四血管闭塞法制作全脑缺血大鼠模型。
Objective to examine whether spreading depression (SD) precondition can induce brain ischemic tolerance.
目的验证扩散性抑制(SD)可诱导脑缺血耐受。
Methods The model of brain ischemic and hypoxic injuries in rats was established by unilateral carotid artery ligature just for 2 h.
方法采用结扎大鼠颈动脉及进入低氧仓的方法建立脑缺血、缺氧损伤模型；
Methods: The manifestations of cerebral lesions and apoptosis in the brain ischemic rats with hyperglycemia were investigated by HE and TUNEL staining.
方法：采用大鼠高血糖全脑缺血模型，用HE和TUNEL染色，对比检测脑细胞损伤和凋亡。
The finding and study of brain-derived erythropoietin(EPO) will provide new insight into mechanisms of brain ischemic tolerance and new inspiration for the prevention and cure of cerebral ischemic.
脑源性EPO的发现和研究，为探讨脑缺血耐受机制提供了新的视角，也为脑缺血的防治提供了新的启示。
They studied the effect of phenylmethylsulfonyl fluoride, a phospholipase C inhibitor, on ischemic injury to brain mitochondria in rats.
他们研究了磷脂酶C抑制剂苯甲基磺酰氟对大鼠脑线粒体缺血性损伤的作用。
Researchers have also found that up to two drinks per day may slash in half the risk for ischemic strokes, the most common type, which occur when arteries that feed blood to the brain become blocked.
研究人员还发现，每天喝两次酒可以使缺血性中风减少一半的发病风险，缺血性中风是最常见一种中风病，当供给大脑血液的动脉发生阻塞时发病。
MRI and doppler ultrasound technology currently allow the researchers insight into slowed perfusion and ischemic injury of the MS brain.
MRI和doppler超声波技术目前允许研究者洞悉医学性脑部的慢扩散和非化学性伤害的机理。
Depression increased the risk of ischemic stroke, in which a blood vessel becomes blocked and cannot send blood to the brain.
沮丧会导致缺血性中风，由于血管堵塞导致血液不能输送到大脑。
Small fibrin thrombi can form in small arteries of brain, heart, lungs, kidneys, and other organs to produce ischemic tissue damage.
小纤维蛋白血栓可在脑、心脏、肺、肾以及其它器官的小动脉中形成，引起组织缺血。
Conclusion Hyperbaric oxygen treatment had an obvious effect on ischemic brain injury, and increasing the expression of Neuroglobin might be one of the possible mechanisms of the therapy.
结论高压氧对缺血性脑损伤有显著治疗作用，提高脑红蛋白的表达水平可能是其治疗的机制之一。
Pathological examination of the brain of the rabbits revealed that there was local ischemic change of the neurons of the cortex and necrosis in the subcortical region in a few of the rabbits.
研究了不同部位脑组织的病理变化，主要改变为大脑皮层神经细胞呈局部缺血性改变，少数皮层下有软化坏死灶。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Conclusion the expressions of STAT3 increased might indicate the transmission course in ischemic nerve cell signal, and play a key role in neuron apoptosis of ischemic brain injury.
结论STAT 3蛋白表达增强可能介导缺血性脑损伤的信号转导过程，其在脑缺血损伤神经元凋亡过程中起关键作用。
AIM To study the protective effects of monosialotetrahexosylganglioside (GM1) after hypoxic ischemic brain damage (HIBD) in neonatal rats.
目的研究外源性单唾液酸四己糖神经节苷脂（GM1）对新生鼠缺氧缺血脑损伤（HIBD）的保护作用。
Objective to explore the influences of early interventions on learning, memory and neuron apoptosis in the brain of neonatal rats with hypoxic ischemic brain damage (HIBD).
目的探讨早期干预对缺氧缺血性脑损伤(HIBD)大鼠学习记忆能力及神经元凋亡的影响。
Through investigating the effect of mild hypothermia on activity of nitric oxide synthase(NOS) in cortical neurons and glycemia levels of neonatal rats with hypoxic ischemic brain damage(HIBD).
通过亚低温对新生鼠缺氧缺血性脑损伤（HIBD）大脑皮质神经元一氧化氮合酶（NOS）及血糖水平影响的研究，探讨亚低温对缺氧缺血性脑损伤的保护作用机制。
Objective To explore the effect of N-acetylcysteine (NAC) on intercellular adhesion molecule-1(ICAM-1) expression and apoptosis in neonatal rats brain after hypoxic and ischemic brain damage (HIBD).
目的探讨N-乙酰半胱氨酸（NAC）对缺氧缺血脑损伤（HIBD）大鼠脑细胞间黏附分子-1及细胞凋亡的影响。
Objective To study the application of ultrasound diagnosis of various brain injury caused by hypoxic-ischemic encephalopathy (HIE).
目的探讨超声诊断在新生儿缺氧缺血性脑病(HIE)各种脑损伤中的应用价值。
Objective To observe the effect of hypothermia on amino acid release in ischemic side of brain in gerbils.
目的观察低温对沙土鼠单侧脑缺血时氨基酸递质释放的影响。
Objective To study the expression of ICAM 1 at gene transcription level in neonatal rats after hypoxic ischemic brain damage(HIBD).
目的研究新生动物缺氧缺血性脑损伤（HIBD）后脑细胞间粘附分子-1（ICAM- 1）在转录水平表达规律。
Objective To investigate the change of cognitive function in the elders with first acute ischemic brain stroke.
目的探讨老年急性缺血性脑卒中患者首次发病后认知功能的改变。
The pathologic mechanism that hyperglycemia intensified the ischemic brain damage was discussed.
作者讨论了高血糖加剧缺血性脑损害的病理机制。
Objective To investigate the changes of blood coagulation state and some prothrombin's function in newborn piglets at different time after hypoxic-ischemic brain damage (HIBD).
目的研究缺氧缺血新生猪凝血状态，以及部分凝血因子促凝活性的动态变化，探讨缺氧缺血性脑损伤（HIBD）时凝血功能变化。
OBJECTIVE: To discuss the protective effects of nimodipine on acute ischemic brain injury caused by activation of phospholipase A2. DESIGN: a completely randomized controlled trial.
目的：探讨尼莫地平对磷脂酶a 2激活致急性缺血性脑损伤中的保护作用。设计：完全随机对照实验。
Hypoxia ischemic brain damage (HIBD), which resulted from neonatal asphyxia, is the main cause of death and disability in neonatal periods.
围产期窒息所致缺氧缺血性脑损伤(HIBD)是新生儿死亡和残疾的主要原因。
The mechanism of ischemic brain injury has not been elucidated thoroughly now, and there is no effective therapy in clinic.
缺血性脑损伤的发生机制目前尚未明确，临床缺乏有效的治疗手段。
Objective To develop an improved neonatal piglet model of hypoxic ischemic brain damage (HIBD).
目的研究新生猪缺氧缺血脑损伤(HIBD)模型的制备。
Objective To develop an improved neonatal piglet model of hypoxic ischemic brain damage (HIBD).
目的研究新生猪缺氧缺血脑损伤(HIBD)模型的制备。
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