氯法拉滨注射液适用于对至少2种治疗方案无效的儿童(1 - 21岁)难治性或复发性急性淋巴细胞白血病的治疗。
Clofarabine injection is indicated for the treatment of pediatric patients (ages 1-21 years) with relapsed or refractory acute lymphoblastic leukemia after at least 2 previous regimens.
研究复发性、难治性中高度恶性非霍奇金淋巴瘤的解救治疗。
To study the salvage treatment of relapsed or refractory intermediate-high aggressive non-Hodgkin lymphoma (NHL).
目的：研究MOD方案对难治性、复发性多发性骨髓瘤(MM)的疗效和毒性反应。
Objective to study the effect and toxicity of MOD regimen in treatment of refractory or recurrent multiple myeloma (MM).
目的：观察eshap方案治疗难治性或复发性非霍奇金淋巴瘤(NHL)的疗效及不良反应。
Objective: to observe the efficacy and the side effect of ESHAP in the treatment of refractory or recurrent Non-Hodgkin's lymphoma (NHL).
背景和目的：难治性急性髓系白血病(aml)包括复发性aml、原发耐药aml、老年aml及继发aml，是目前白血病治疗困难的类型。
Background and objective: Relapse AML, refractory AML, AML in the old and secondary AML are all AML subtypes hard to cure.
由于该病的难治性和易复发性等特点，目前提倡建立社会-心理-生物医学模式，采用心理和药物治疗相结合的治疗方式。
Because the disease is refractory and recurrent, now it is promoted to establish the social-psychological-biomedical model, adopting a combination treatment of psychological and drug therapy.
替匹法尼用一周，停一周剂量递增治疗复发性，难治性或高危髓细胞性白血病1阶段试验。
A phase 1 trial dose-escalation study of tipifarnib on a week-on, week-off schedule in relapsed, refractory or high-risk myeloid leukemia.
我们在此报道一个II期多中心单一研究项试验，以评价来那度胺口服单一疗法治疗复发性或难治性侵袭性NHL的安全性和有效性。
We report the results of a phase II, single-arm, multicenter trial evaluating the safety and efficacy of lenalidomide oral monotherapy in patients with relapsed or refractory aggressive NHL.
目的观察131I-GMCSF诱导HL60/ADM细胞凋亡，探讨其诱导HL60/ADM细胞凋亡的机制，比较131I-GMCSF对HL60/ADM细胞和HL60细胞的细胞毒作用，为其临床治疗难治性或复发性急性髓细胞性白血病提供实验依据。
Objective: To compare the cytotoxicity of HL60/ADM cells and HL60 cells treated with 131I-GMCSF and to investigate the mechanism of the apoptosis of HL-60/ADM cells induced by 131I-GMCSF.
目的观察131I-GMCSF诱导HL60/ADM细胞凋亡，探讨其诱导HL60/ADM细胞凋亡的机制，比较131I-GMCSF对HL60/ADM细胞和HL60细胞的细胞毒作用，为其临床治疗难治性或复发性急性髓细胞性白血病提供实验依据。
Objective: To compare the cytotoxicity of HL60/ADM cells and HL60 cells treated with 131I-GMCSF and to investigate the mechanism of the apoptosis of HL-60/ADM cells induced by 131I-GMCSF.
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