研究小组通过遗传工程改造小鼠使其缺乏TEX11功能，结果发现TEX11功能的缺失导致在配子形成过程中染色体不联会。
The team genetically engineered male mice such that they lacked TEX11 function and found that this caused chromosomal asynapsis during the process of gamete formation.
他们从发生实验性IRI的小鼠身上分离了白细胞，并注入遗传工程摘除了胸腺（从而不能产生T淋巴细胞）的小鼠，实验以未注射的同样小鼠为对照。
These cells were injected into mice engineered without a thymus gland, which produces T cells. A comparison group of genetically engineered mice got no injections.
他们从发生实验性IRI的小鼠身上分离了白细胞，并注入遗传工程摘除了胸腺（从而不能产生T淋巴细胞）的小鼠，实验以未注射的同样小鼠为对照。
These cells were injected into mice engineered without a thymus gland, which produces T cells. A comparison group of genetically engineered mice got no injections.
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