表明FMCM对照射造成的造血损伤具有良好的保护和治疗作用。
The above results suggest that FMCM has good protective and promoting effects on reducing hematopoietic damage caused by irradiation.
结论：四物汤可以在一定程度上逆转综合放血致小鼠的造血损伤。
Conclusion: Siwu decoction significantly improve the hematopoiesis in the mice with blood deficiency induced by compound method mentioned above.
电离辐射在工业、医学的广泛应用可造成机体严重造血损伤，造血干细胞移植是救治的重要措施。
The application of ionizing radiation to industry, medicine may lead to severe hematopoietic injuries, while hematopoietic stem cells transplantation is the important therapy.
目的探讨归芪口服液对放射损伤小鼠骨髓组织中CD54表达的影响及其促进早期骨髓造血恢复的可能机制。
Objective To explore the effect of Gui Qi oral liquor on the expression of CD54 in bone marrow and its possible mechanisms of hematopoietic recovery in acute irradiation injured mice.
目的：进一步探讨外周血干细胞移植后患者骨髓造血微环境损伤的机理。
Objective: to investigate the injury mechanism of bone marrow hematopoietic microenvironment from PBSCT patients anterior and post pretreatment.
目的作者自1995 ~ 2002年采用人造血管移植修复四肢大血管损伤23例，进行回顾性研究人造血管在治疗四肢大血管损伤中的应用。
Objective to retrospectively study the results of 23 cases with major vascular injuries in extremities by artificial blood vessel graft from 1995 to 2002 in our department.
结论：口服减毒沙门氏菌sl 3261为载体的PF 4基因可以保护小鼠免受损伤，并促进化疗损伤小鼠的造血恢复。
Conclusions: Oral attenuated Salmonella Typhmuriumas SL3261 transfected with PF4 gene can protect mice from injury and promote the hematopoiesis reconstitution of chemotherapy injured mice.
辐射及某些抗癌药物会损伤骨髓，损害免疫力。检查骨髓有助于诊断有关血液及造血系统的疾病。
Radiation and some anticancer drugs can damage marrow and impair immunity. Bone-marrow examination helps diagnose diseases related to blood and blood-forming organs.
目的探讨人造血管移植术修复四肢血管损伤患者的护理方法。
Objective To explore the nursing methods on patients with major vascular injury in extremities treated by artificial blood vessel graft.
方法以免疫学和血液学常规方法观察PF对机体免疫功能和辐射所致造血功能损伤的影响。
Methods The influence of PF on immune function and radiation-induced hematopoietic injury was observed by using common methods in immunology and hematology.
关于造血功能损伤与修复的研究与应用，既往多侧重于造血实质细胞，对构成造血微环境重要成分的造血基质细胞尚需深入研究。
The research and application on injury and repair of hematopoietic function in the past focus more on HSC, and it is needed to further study of the stromal cells.
结论：肽hp - 6可刺激造血系统相关细胞增殖，对小鼠造血功能与抵抗化疗药物损伤有一定的增强作用。
Conclusion: Peptide HP-6 could promote the proliferation of cells related to hematopoietic system, enhance mouse hemopoiesis function and the resistance to chemotherapeutic injury.
所述细胞可用于造血干细胞移植以及使用干细胞移植修复损伤组织等。
The cells are useful for hematopoietic stem cell transplantation, repair of an injured tissue utilizing stem cell transplantation, or the like.
辐射及某些抗癌药物会损伤骨髓，损害免疫力。检查骨髓有助于诊断有关血液及造血系统的疾。
Radiation and some anticancer drugs can damage marrow and impair immunity. bone - marrow examination helps diagnose diseases related to blood and blood-forming organs.
目的研究新型抗放药e838对放烧复合伤和单纯放射损伤造血功能的防护效果。
Objective to compare the effects of the radiation protective agent E838 on hematopoiesis of mice with simple irradiation and combined radiation with burn injury.
目的研究旋转恒定磁场对5-氟尿嘧啶（5-FU）损伤小鼠造血功能的保护作用。
ObjectiveTo study the hemoprotective effects of rotating and stationary magnetic field(RSMF) in mice treated with 5-Fluorouracil(5-FU).
结论低浓度苯可引起小鼠造血系统损伤、过氧化损伤及致突变作用。
Conclusions It suggested that low concentration benzene results in hazards of histopathological system and could develop lipid peroxidation and mutagenicity.
辐射后，小鼠骨髓中VEGF的改变与骨髓基质的损伤、造血恢复的关系值得进一步研究。
The association among expression of VEGF, hematopoietic microenvironment and hematopoietic reconstitution deserve fur-ther study.
结论：采用人造血管移植修复四肢主要血管损伤是保住肢体及其功能的方法之一，其优势为术后短期评估血管通畅率及肢体功能优良率均较好。
CONCLUSION: vascular prosthesis grafting is the one of the methods for repairing the major vascular injuries so as to preserve the extremities and their functions.
目的探讨放烧复合伤对骨髓造血微环境损伤的机制。
Aim to investigate the mechanism of the radiation burn combined injury to the bone marrow hematopoietic microenvironment in the mice.
目的探讨放烧复合伤对骨髓造血微环境损伤的机制。
Aim to investigate the mechanism of the radiation burn combined injury to the bone marrow hematopoietic microenvironment in the mice.
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