研究背景：在多发性硬化的病变过程中通过多种细胞毒效应产生过氧化氮阴离子。
Background: Peroxynitrite was hypothesized to be involved in the pathogenesis of multiple sclerosis through its various neurotoxic effects.
方法以小鼠血清中谷胱甘肽过氧化物酶（GSH-PX）及一氧化氮（NO）为指标进行实验观察。
Method Using the serum levels of glutathione-peroxidase (GSH-PX) and nitric oxide(NO) as the indices to prove the medicine's curative effect.
结论2 -乙氧基乙醇能引起肝脏一氧化氮自由基增加并诱发脂质过氧化反应对肝脏发生毒性作用。
Conclusion the study demonstrated that 2 ethoxyethanol can increase the liver level of no free radicals and the lipid peroxidation may play an important role in 2 ethoxyethanol's liver toxicity.
方法应用过氧化氢损伤培养的人脐静脉内皮细胞，并与不同浓度乙醇进行孵育，检测内皮细胞产生的内皮素及一氧化氮。
Methods Impaired by hydrogen peroxide, cultured endothelial cells intubated with different concentration of alcohol in order to determine the production of et and NO.
目的建立拟血管性痴呆的动物模型，探讨针刺对VD大鼠模型的学习记忆行为及脑内谷胱甘肽过氧化物酶（GSH-PX）及一氧化氮（NO）含量的影响，观察海马神经元细胞凋亡情况。
Purpose To study the effect of acupuncture on the learning dysmnesia of VD rats activity of GSH-PX and content of NO and to observe injured neuron in hippocampal region.
方法：建立四氧嘧啶糖尿病小鼠动物模型，测定肝脏抗氧化酶、ATP酶活性、脂质过氧化产物、一氧化氮(NO)和肝糖原含量。
METHODS: the model of alloxan diabetic mice was established to determined the activities of enzymes of antioxidant and ATP, and the contents of MDA, NO and glycogenesis.
方法：建立四氧嘧啶糖尿病小鼠动物模型，测定肝脏抗氧化酶、ATP酶活性、脂质过氧化产物、一氧化氮(NO)和肝糖原含量。
METHODS: the model of alloxan diabetic mice was established to determined the activities of enzymes of antioxidant and ATP, and the contents of MDA, NO and glycogenesis.
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