结论AT P不仅抑制人胃癌细胞的增殖，而且诱导分化，使人胃癌细胞的恶性表型向正常表型逆转。
Conclusion ATP not only inhibits proliferation but also induces differentiation of a human gastric cancer cell line with reversion from malignant toward normal phenotype.
我们假设这样一种复杂的表型逆转可能是由于多重信号因子（彼此结合时可允许肿瘤细胞形成静态类腺泡结构）的作用。
We hypothesized that such a complex phenotypical reversion is likely the result of multiple signaling factors that in combination allow cancer cells to form quiescent acinar-like structures.
对HSC起源、分布与表型转变的研究将为治疗与逆转肝纤维化提供新的方向。
The study of HSC on the origin, distribution and phenotypic changes will provide a new direction for the treatment and reversal of hepatic fibrosis.
目的研究EB病毒LMP1反义基因对人鼻咽癌CNE2细胞恶性表型的逆转作用。
Objective To study the reverse effect on the malignant phenotype of human NPC cells CNE2 transfected with EBV-LMP1 antisense gene.
结论MDR1可能参与胰腺癌细胞株化疗耐药；中等剂量的维拉·帕米联合化疗可逆转具有MDR1表型的人胰腺癌细胞株对adm和MMC的耐药现象。
Conclusions Moderate dose of verapamil can reverse chemoresistance of pancreatic carcinoma cell line to ADM and MMC that might be related to MDR1.
以上实验进一步说明鼻咽癌是多基因改变的疾病，NAG1 2的重表达有助于鼻咽癌恶性表型的逆转。
These data further demonstrated that NPC is a disease involving polygenic alteration and NAG12 gene could favor malignant phenotype reversion of NPC cells.
以上实验进一步说明鼻咽癌是多基因改变的疾病，NAG1 2的重表达有助于鼻咽癌恶性表型的逆转。
These data further demonstrated that NPC is a disease involving polygenic alteration and NAG12 gene could favor malignant phenotype reversion of NPC cells.
应用推荐