TMV还可以诱导血小板血栓恶烷A_2的形成。
The TMV also induced the formation of Thromboxane A2 (TXA2) of platelets.
冠心病患者血小板PKB高活化性与血小板异常活化及血小板血栓形成有关。
The results suggest that the activated activities for platelet PKB was the causes of unusual activated platelet and platelet thrombosis in coronary heart disease.
血栓调节蛋白可完全抑制凝血酶和利斯托司汀诱导的血小板聚集。
Thrombomodulin can entirely inhibit thrombin and ristocetin-induced platelet aggregation.
此外，血小板凝固不但会在心瓣上形成微小血栓，致使栓体不断增长；还会引起血管发炎，从而导致血液无法输送到心脏和大脑。
In addition, platelet clumping can cause small blood clots, growths on the heart valves or inflammation of blood vessels that can block the blood supply to the heart and brain.
研究者们正在寻求一种能够更温和地抑制血小板的治疗方式，血小板是血液中盘状的物质，能形成血栓。
Researchers are seeking treatments that more gently orchestrate activity of platelets, disk-shaped particles in the blood that form clots.
也许最常见的血小板功能缺陷是使用阿司匹林造成的不可逆的血栓素抑制(这是血小板聚集所必要的)。
Probably the most common platelet function defect is the irreversible inhibition of thromboxane (which is necessary for platelet aggregation) caused by aspirin administration.
阿司匹林除了具有止痛效果，还可以防止血小板互相粘连，从而让血管变细。它还可以降低由血栓引起的心脏病和中风的危险。
Beyond its painkilling effects, aspirin prevents blood platelets sticking together and so thins the blood. This reduces the risk of blood clots that can cause heart attacks and stroke.
目的：观察舒心饮与抗血小板药物联合应用对冠状动脉支架血栓等术后冠脉事件的预防作用。
Objective: To evaluate the preventive effect of antiplatelet regimens and it′s combination with Shuxinyin (SXY) on coronary events including stent thrombosis after stenting implantation.
血纤维蛋白和血小板一起形成血栓。血友病就是因为遗传上缺乏一种凝血胶原引起的。
Fibrin and platelets combine to form a clot. Hemophilia is caused by a hereditary lack of one of the clotting factors.
因为血栓形成和血小板聚集是相对紧急的事情，所以阿斯匹林的对血管疾病的好处在五年内就可以观察的到并不奇怪。
Because thrombosis and platelet aggregation are relatively acute events, it is not surprising that the benefit of aspirin for vascular disease was observed within five years, the investigators said.
结论：云南白药能增加血小板活化，促进局部止血，但它不影响凝血物质和D -二聚体的含量，不增加血栓形成的危险。
Conclusion: Yunnan Paiyao can increase platelet activating, accelerate local hemostasis, but has no influence on prothrombin and D-dimer content, thus will not increase the dangerous of thrombosis.
抗血小板药物(例如波利维)被用来帮助预防伴心脏病人群中潜在有危险性的血栓形成。
Antiplatelet drugs, such as Plavix, are used to help prevent potentially dangerous blood clots from forming in people with heart disease.
因此抗凝剂对于防治静脉血栓栓塞十分有效，而血小板功能抑制药的疗效则差一些。
Anticoagulants are therefore very effective for prevention and treatment of venous thromboembolism, and drugs that suppress platelet function are of less benefit.
受伤以后，血小板粘在破损血管壁上，并且互相粘结，从而使蛋白质凝固，形成血栓。
After an injury, platelets stick to the walls of damaged vessels, to each other, and to clotting proteins, forming a plug.
结论调脂胶囊可通过延长血浆中aptt、PT、TT，影响PAG，改善血小板的功能，防止血栓的形成，以发挥抗动脉粥样硬化的作用。
Conclusion: Tiaozhi Capsule can prolong APTT, PT, TT, and influence PAG, and improve the function of the platelet and prevent the formation of thrombus to cure the AS.
尽管普遍认为抗凝疗法可预防静脉血栓形成，双重抗血小板疗法仍可用来治疗适应性较差的患者。
Although it is presumed that prevention of venous thrombosis is best achieved with anticoagulation, dual antiplatelet therapy can be a substitute for patients with poor compliance.
现在专家的一致意见认为，这可能是由于抗血小板和抗血栓治疗不足所引起的。
And there's consensus that the increased ischemic events are related to insufficient antiplatelet and antithrombotic therapy.
另6例JAK2V617F点突变阴性患者仅1例有血栓史，血小板数目及骨髓巨核细胞数目相对较低。
Out of other 6 IMF patients without JAK2V617F point mutation only 1 patient had thrombosis, and lower counts of platelets in perepheral blood and megakaryocytes in bone marrow.
大鼠血栓湿重降低；抑制兔血小板的聚集率。
It also decreased the rats' thrombosis and inhibited the rabbits' platelet aggregation rate.
还具有改善微循环、抗血小板的聚集和血栓形成，并能使血液粘度下降，改善血液循环。
And have improvement microcirculation, resist blood platelet aggregation and thrombopoiesis, can make blood viscosity come down, improve blood circulation.
观察20例急性脑梗塞，30例冠心病在微循环中微血栓的变化，同时检测血小板聚集率、D-2聚体。
The microthrombi, D- Dimer and platelet aggregation rate were observed in 30 patients with coronary heart disease and 20 patients with acute cerebral infarction.
目的观察海洋假单胞菌碱性蛋白酶（MPAP）的溶栓和抑制血栓形成的作用，研究其对纤溶、凝血和血小板聚集功能的影响。
ObjectiverTo observe the thrombolysis and anti-thrombosis effects of MPAP; To study the actions of MPAP on blood coagulation, platelet aggregation and fibrinolytic activity.
当正常血小板暴露于血管内属下的结构中时，几秒钟内即导致血栓的形成。
Exposure of normal platelets to vascular subendothelial structures results, within a matter of seconds, in the formation of a haemostatic plug.
它能激活血小板的活性因子，对抗血液凝固，能有效预防脑血栓、脑梗塞、心肌梗塞等心脑血管疾病。
Can excite the active factor of the blood platelet, oppose the blood to solidify, play a role in preventing the coronary heart disease and brain blood vessel pathological change.
作者研究了该类止血材料对血小板粘附及体外血栓形成的影响。
In this paper, the effect of these materials on platelet and thrombus formation is studied.
斑块破裂提供了血小板聚集和血栓形成的场所，导致不稳定性心绞痛或心肌梗死。
Plaque rupture provides a focus for platelet deposition and thrombosis, and results in unstable angina or myocardial infarction.
方法：采用全蝎提取液给家兔、小鼠静脉注射，并测定其血小板最大聚集率、血栓重量、纤维蛋白原含量及优球蛋白溶解时间的变化。
Methods: the changing of platelet aggregation rate, the thrombus weight, the content of fibrinogen and the euglobulin lysis time were determined after SEF iv on rabbit and mice.
方法：采用全蝎提取液给家兔、小鼠静脉注射，并测定其血小板最大聚集率、血栓重量、纤维蛋白原含量及优球蛋白溶解时间的变化。
Methods: the changing of platelet aggregation rate, the thrombus weight, the content of fibrinogen and the euglobulin lysis time were determined after SEF iv on rabbit and mice.
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