目的分析小鼠螺旋神经节细胞电压依赖性钙通道亚单位的类型。
Objective To investigate subunit type of voltage-dependent calcium channels in the spiral ganglion cells of the mouse.
作为内耳螺旋神经节细胞基因转染载体的可行性及其转染特点。
To study the feasibility and the characteristics of recombinant baculovirus as spiral ganglion cells (SGC) gene transfer vector.
结论：耳蜗螺旋神经节神经元在体外有稳定的可塑性及轴突再生修复能力。
Conclusion: The spiral ganglion neurons have stable plasticity and ability of neuritic regeneration.
结果：GAP-43、NGF在实验动物耳蜗螺旋神经节细胞中都有一定的表达；
Results:GAP-43 and NGF had certain expression in the spiral ganglion cells of cochlea of the tested animals.
目的：在体外建立大鼠耳蜗螺旋神经节神经元培养模型，并提高其产出率及细胞活性。
To establish a cytological model of spiral ganglion neurons in vitro and to improve cell yield and viability.
目的：探讨脑细胞生长肽是否有预防庆大霉素对豚鼠耳蜗螺旋神经节细胞的损害的作用。
AIM: to investigate the effects of growth peptide of cerebral cells in preventing damage of gentamicin to cochlear spiral ganglion cells (SCGs) in ototoxic guinea pigs.
结果：显示耳蜗螺旋神经节神经元在体外无血清培养条件下，可以存活并进行正常分化。
Results: The spiral ganglion neurons could survive and had a normal phenotypic differentiation in serum-free medium.
结论：金匮肾气丸可提高庆大霉素致聋豚鼠耳蜗gap - 43、NGF表达，对螺旋神经节细胞进行修复。
Conclusions: Jinkui Shenqi Pills can restore the injured spiral ganglion cells by increasing the expression of GAP-43 and NGF in the cochlea of gentamicin-induced deafness guinea pigs.
目的：建立稳定表达nt3的NIH3T3细胞株，通过体外共培养实验观察其对耳蜗螺旋神经节细胞生长的影响。
AIM: to establish a NIH3T3 cell line which expresses human NT3 and to observe the growth of cochlear ganglion cells by co culturing.
结果：将卡铂耳中毒灰鼠的耳蜗标本与正常灰鼠进行比较，显示内毛细胞，神经纤维和螺旋神经节的损失百分比基本一致。
Results: Compared to normal ears, the carboplatin treated ears showed missing hair cells, nerve fibers and spiral ganglion cells, with the same percentage of loss at different levels.
目的：观察水杨酸钠给药对幼年豚鼠螺旋神经节谷氨酸脱羧酶（GAD）蛋白表达和听性脑干诱发电位（ABR）阈值的影响。
Objective:To study the effects of sodium salicylate on the expression of GAD protein and ABR threshold in spiral ganglion of juvenile guinea pigs.
结果 ：豁痰祛瘀颗粒能降低糖尿病大鼠升高的血糖，减轻糖尿病大鼠早期耳蜗基底膜、血管纹增厚程度，使稀疏的螺旋神经节细胞变致密。
Result : GEPRB can reduce the cumulating thickness of the basilar membrane and that of the vascular pattern, so as to tighten up the sparsity of the spiral ganglion cells.
结果 ：豁痰祛瘀颗粒能降低糖尿病大鼠升高的血糖，减轻糖尿病大鼠早期耳蜗基底膜、血管纹增厚程度，使稀疏的螺旋神经节细胞变致密。
Result : GEPRB can reduce the cumulating thickness of the basilar membrane and that of the vascular pattern, so as to tighten up the sparsity of the spiral ganglion cells.
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