氧化损伤，或氧化应激，出现在被称为自由基的高活性分子攻击和破坏细胞蛋白质，脂类(脂肪)和脱氧核糖核酸时。
Oxidant injury, or oxidative stress, occurs when highly reactive molecules called free radicals attack and damage cellular proteins, lipids (fats) and DNA.
结论热休克蛋白具有保护肾脏抗氧化酶活性的作用，热预处理能够减轻严重烧伤后肾损伤。
Conclusion Heat shock precondition can protect the activity of antioxidase in rat kidney, and decrease the injury of kidney after severe burn.
在活细胞中，藻蓝蛋白既可以作为蛋白存储单位，也可以作为抗氧化剂，使细胞在一定限度内免受光损伤。
In the living algae cell, phycocyanin can serve as a protein storage unit and as an antioxidant, protecting the cell from certain wavelengths light damage.
目的探讨在紫外线诱导的人晶状体上皮细胞DNA损伤修复过程中端粒酶活性和氧化损伤蛋白的变化和作用。
Objective To investigate the changes and the role of telomerase activity and other stress-related proteins in the process of UV-induced DNA damage and repair in human lens epithelial cells.
活性氧在生物大分子如DNA、蛋白质等氧化性损伤中起着非常重要的作用。
Active oxygen has an important role in the oxidative damage of biomacromolecules for example DNA, protein and so on.
ROS通过氧化线粒体心磷脂、线粒体dna和线粒体重要的蛋白质对线粒体造成氧化损伤，进而诱导细胞凋亡。
Through oxidizing the mitochondrial cardiolipin, mitochondrial DNA and important proteins in mitochondria, ROS leads to the oxidative stress towards mitochondria, further induces the cell apoptosis.
结论模拟失重可诱导PC 12细胞NO的合成，增加蛋白质氧化损伤程度。
Conclusions Simulated weightlessness may induce the (over) production of NO, and resulted in increased protein oxidative damage in PC12 cells.
研究了SO2衍生物对小鼠脾、胃蛋白质的氧化损伤作用并探讨其分子作用机制。
The oxidative damage action of SO2 derivatives on the mice spleen and stomach proteins was studied, and its molecular action mechanism was probed into.
结论线粒体型硫氧还蛋白还原酶基因和蛋白表达的增加有利于大鼠心肌抗氧化损伤。
Conclusions the increased expression of mitochondria thioredoxin reductase may protect the heart against oxidative stress injury.
采用氧化前处理与蛋白酶及TG酶三者结合新工艺，可以在保证纤维损伤小的前提下，提高织物的抗毡缩性。
The wool anti-felting performance could be improved and the wool fiber damage could be controlled by use of new bio-finishing process of the wool combining oxidation, protease and TG enzyme.
表明富含腺嘌呤类衍生物的玉米幼芽提取物对体外培养的小鼠皮肤成纤维细胞具有抗凋亡保护作用，在细胞氧化损伤过程中，对细胞膜蛋白和结构性蛋白有保护作用。
Above all, extracts of maize plumule rich in adenine ramification could prevent MDF from apoptosis, and protect the structure protein and membrane protein when the oxidative damage happened.
目的观察不同浓度牛磺酸对脂质过氧化损伤的红细胞以及自然衰老红细胞的抗氧化酶类和膜骨架蛋白保护作用，以探讨其抗氧化和抗衰老的作用机制。
Objective To observe the effect of taurine (Tau) on erythrocyte lipid peroxidative in-jury and erythrocyte survival, and to explore the protective mechanism of Tau on erythrocyte.
结论晚期氧化蛋白产物能通过氧化应激引起血管内皮细胞损伤。
Conclusion AOPP decreased endothelial cell viability via induction of oxidative stress.
结论：氯通道阻断剂NPPB或NFA能够在一定程度上减轻氧化应激引起的C6细胞损伤，可能与调节细胞膜功能和下调CLIC 4蛋白表达有关。
CONCLUSION: the chloride channel blockers NPPB or NFA lessen the oxidative injury of C6 cells through modulating the function of membrane and down-regulating the protein expression of CLIC4.
获得的星虫蛋白多肽在动物实验中表现出具有抗氧化，增强运动能力及防止运动造成的氧化 损伤。
The obtained sipunculid polypeptide shows activity of resisting oxidation, enhancing sports ability and preventing oxidation injury caused by sports in animal experiments.
应用一氧化氮和基质蛋白酶的抑制剂明显降低肝窦内皮细胞的损伤，表明一氧化氮和基质蛋白酶在缺血-再灌注损伤中是重要的介导物。
The inhibitor for NO or for MMPs significantly reduced the LSEC IRI, suggesting that NO and MMPs are important mediators in the LSEC IRI.
应用一氧化氮和基质蛋白酶的抑制剂明显降低肝窦内皮细胞的损伤，表明一氧化氮和基质蛋白酶在缺血-再灌注损伤中是重要的介导物。
The inhibitor for NO or for MMPs significantly reduced the LSEC IRI, suggesting that NO and MMPs are important mediators in the LSEC IRI.
应用推荐