上述观察结果为阐明自体抗原的半抗原化为何会引起自身免疫性疾病提供了一种似乎合理的解释。
The observed results provide a plausible mechanism for how haptenization of self-antigens can lead to the development of autoimmunity.
由这些结果，我们推论在活体动物内，抗登革病毒且具交叉反应的自体抗体与自体抗原的结合，会导致细胞功能的失常或破坏。
We conclude that the anti-dengue antibodies that cross-react to autoantigen will cause target cell to damage or function loss.
竞争结合的实验分析指出SARS冠状病毒棘蛋白区域2 （S2） 确实有交互作用的抗原决定位存在，进一步的实验鑑定出数个自体抗原。
Preabsorption and binding assays indicated the existence of cross-reactive epitopes on SARS-CoV spike protein domain 2 (S2). Several candidate autoantigens have been identified.
结论特定的抗原激活自体免疫系统产生了相应的抗体从而达到治疗效果。
Conclusion the specific antigen activates the immune system to produce the corresponding antibody to achieve the therapeutic effect.
该细胞接触肝癌细胞抗原后刺激自体淋巴细胞增殖的能力增强。
The ability of stimulation autologous lymphocytes proliferation of cells were better than original cells.
通过对进程行为的检测来提呈抗原，进而定义了自体，并给出了一个基于免疫细胞机制的检测算法来检测未知病毒。
The method presentsantigens and defines self by monitoring the behaviors of running processes, and uses an immune algorithm based on immune cells to detect any possible viruses.
目的经脱抗原处理的牙本质（HPDD）植入治疗萎缩性鼻炎替代自体骨移植。
Objective To treat atrophic rhinitis by implanting homologue of partly beantigenic dentinum (HPDD) instead of the autogenic bone implantation.
结果自体角质形成细胞可抑制淋巴细胞对同种异体抗原的增殖。
Methods The mixed epidermal cell and lymphocyte culture (MELC) was established, in which auto-keratinocytes were added to mimic the effect exerted by the auto-islets in vivo.
摘 要：目的： 探讨利用癌症患者自体成纤维细胞作为抗原提呈细胞，诱导抗肿瘤的细胞毒T淋巴细胞（CTL）反应的可能性。
Objective: To characterize and develop a potential of activating tumor-specific cytotoxic T lymphocyte(CTL) responses with autologous fibroblasts of cancer patients.
使用这款产品，您可以在无自体同源抗原表达的细胞和抗原的情况下，扩增抗原特异性初始cd 4 +和CD 8 +T细胞。
With this product you can expand naive and antigen-specific CD4 + and CD8 + t cells without autologous antigen-presenting cells and antigen.
再将RB肿瘤抗原致敏树突状细胞，与自体淋巴细胞共孵育后，能诱导出特异性杀伤肿瘤细胞的CTL。
The dendritic cells were induced by RB tumor antigen and then it cooperated with RB tumor lysates in vitro to induce antigen specific CTL which can kill the SO-RB50 target cells specifically.
再将RB肿瘤抗原致敏树突状细胞，与自体淋巴细胞共孵育后，能诱导出特异性杀伤肿瘤细胞的CTL。
The dendritic cells were induced by RB tumor antigen and then it cooperated with RB tumor lysates in vitro to induce antigen specific CTL which can kill the SO-RB50 target cells specifically.
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