患者呈现腹水及明显的脾肿大。
脾肿大几乎是经常存在的。
主要的临床症状是贫血，脾肿大和黄疸。
The major clinical features are anemia, splenomegaly, and jaundice.
临床以发热、贫血、肝脾肿大为主要表现。
Main clinical manifestations include fever, anemia and hepatosplenomegaly.
脾肿大常见原因之一是肝硬化门静脉高压。
One of the most common causes for splenomegaly is portal hypertension with cirrhosis of the liver.
肝炎病毒带原、肝脓疡、肝脾肿大、黄疸。
Hepatitis virus carrier, hepatic abscess, hepatosplenomegaly or icterus.
早期出现肝脾肿大，但淋巴结压痛确属罕见。
Enlargement of the liver and spleen occurs early, but rarely are lymph nodes tender.
多见有肝脾肿大。
病人常常会产生贫血、脾肿大和全身虚弱等症状。
Patients often have anemia, spleen enlargement, and general weakness.
据说大约三分之一或一半以上的病例有栓塞和脾肿大。
Enlargement of the spleen is said to occur in about one-third of cases and embolization, in more than half of cases.
结论中药离子透入对慢性肝炎伴脾肿大的治疗有显著效果。
Conclusion TCM iontophoresis has obvious therapeutic effect on chronic hepatitis with splenomegaly.
目的：研究肝硬化脾肿大及原发性脾肿大患者血流动力学变化。
Objective: To study the hemodynamic changes of patients with cirrhotic splenomegaly and primary splenomegaly.
病人常常会产生贫血、脾肿大和全身虚弱等症状。并发症可致命。
Patients often have anemia, spleen enlargement, and general weakness. Complications can be fatal.
新生儿期先天梅毒症状和体征主要有皮疹、肝脾肿大、肺部感染。
Clinical signs and symptoms of these cases included skin rashes, pulmonary infection, enlargement of liver and spleen and blood abnormalities.
具体评价内容：胁肋疼痛、舌象、肝肿大、脾肿大、肝掌蜘蛛痣。
Specific evaluations: Pain in ribs, tongue demonstration, hepatauxe, enlarged spleen, spider telangiectasia in liver palm.
主要临床表现为长期发热、纳差、消瘦、左上腹痛，脾肿大伴贫血。
The main clinical features included longterm fever, anepithymia, weight loss, pain of left upper quadrant, splenomegalia, and anemia.
多中心CD也可有内脏淋巴组织受累，导致肝脾肿大或其它器官肿大。
Multicentric CD can also affect lymphoid tissue of internal organs, causing the liver, spleen, or other organs to enlarge.
脾静脉结扎后可以成功建立脾肿大和血小板减少、红细胞减少的脾亢模型。
Models of thrombocytopenia, erythropenia and splenomegaly were established successfully by ligation of splenic veins.
这样的脾肿大通常预示有骨髓增生型疾病，例如慢性髓性白血病或骨髓纤维化。
Such massive splenomegaly is usually indicative of some myeloproliferative disease such as chronic myelogenous leukemia or myelofibrosis.
孤立性胃底静脉曲张、肝功能正常、脾肿大是胰源性门脉高压最典型的临床表现。
The major clinical symptoms of pancreatic portal hypertension are isolated gastric fundus varices with normal liver function and unexplained splenomegaly.
临床主要表现为淋巴结肿大、发热、肝脾肿大、皮疹、血沉快和多克隆免疫球蛋白血症。
Main manifestation of the clinical symptoms were enlargement of lymph nodes, fever, hepatosplenomegaly, skin rash, rapid red cell sedimentation rate and polyclonal hyperglobulinemia.
白细胞数低可能是骨髓疾病或脾肿大症状，有些HIV感染病人也会出现白细胞数降低的情况。
Low white counts can be a sign of bone marrow diseases or an enlarged spleen. Low WBC is also found in HIV infection in some cases.
结果9例患者中，4例完全无临床症状，2例有轻度贫血、脾肿大，3例有轻度至中度巩膜黄染。
Results Out of the 9 patients, 4 had no clinical symptoms, 2 showed mild anemia and splenomegaly, 3 were jaundice.
静息性的肝硬化可能在出现因门脉高压引起的充血性脾肿大导致的无症状的血小板减少症之后才被发现。
Silent cirrhosis may be discovered after the finding of asymptomatic thrombocytopenia caused by the congestive splenomegaly of portal hypertension .
持续性原因不明的广泛淋巴结肿大（淋巴结直径大于1厘米），常见于颈、腋及枕部等，以及肝、脾肿大。
Persistent unexplained extensive lymph nodes (lymph nodes greater than 1 cm in diameter), common in the neck, armpit and the occipital, as well as liver and splenomegaly.
新生儿中最常见的临床表现是呼吸窘迫综合症，其次是早产、败血症(均为41%)和肝脾肿大(13%)。
The most common presentations in neonates were respiratory distress syndrome, followed by preterm birth and signs of sepsis (both 41%) and hepatosplenomegaly (13%).
结节型侵犯伴脾肿大者显著高于其他型，结节型者主要为BNHLBMI，表明NHLBMI伴脾肿大与淋巴瘤表型有关。
The frequency of splenomegaly in nodular type NHLBMI was significantly higher than that in any other type. Nodular type NHLBMI occurred mainly in B cell lymphoma.
脾肿大及脾功能亢进症状得到明显缓解，相关实验室检查包括：血小板、血清总胆红素、血清蛋白、PT、SGPT等均有明显恢复。
Symptoms of splenomegaly and hypersplenism were obviously relieved. Relevant laboratory detections revealed distinct redintegration of platelets, common serous bilirubin, serous protein, PT and SGPT.
脾肿大及脾功能亢进症状得到明显缓解，相关实验室检查包括：血小板、血清总胆红素、血清蛋白、PT、SGPT等均有明显恢复。
Symptoms of splenomegaly and hypersplenism were obviously relieved. Relevant laboratory detections revealed distinct redintegration of platelets, common serous bilirubin, serous protein, PT and SGPT.
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