凋亡细胞脾胃湿热组和脾气虚组均高于正常对照组。
The apoptotic cells in both the SSDH and SQD groups were higher than those in the control group.
肝脏中葡萄糖激酶基因表达情况：番泻叶泻下脾气虚组大鼠肝脏中葡萄糖激酶鑫因表达与生理盐水对照组比较显著降低（P<0 .01）。
Compared with the Saline control groups, the gene expression of glucokinase in the liver on model rats of the senna leaf laxation group remarkably decrease (P<0. 01).
结论脾气虚证发生有免疫相关基因组学基础，脾虚时机体免疫功能紊乱。
ConclusionThe genesis of Pi-Qi deficiency syndrome has its immune associated genomic basis, and the immune functions are disordered in patients with that syndrome.
以30例慢性胃病脾气虚患者为实验组，观察其外周血T淋巴细胞亚群的改变，并以60例健康人为对照。
The changes of T lymphocyte subpopulation of peripheral blood were observed in 30 cases of chronic gastropathy with spleen deficiency(Experimental Group, G 1), and in 60 normal cases as control(G2).
以30例慢性胃病脾气虚患者为实验组，观察其外周血T淋巴细胞亚群的改变，并以60例健康人为对照。
The changes of T lymphocyte subpopulation of peripheral blood were observed in 30 cases of chronic gastropathy with spleen deficiency(Experimental Group, G 1), and in 60 normal cases as control(G2).
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