目的研究高血压大鼠在脑缺血损伤时的病理改变。
Objective to investigate the histopathology changes in hypertensive rats after cerebral ischemia.
背景与目的脑缺血损伤后细胞凋亡受诸多基因调控。
Background and objective: Many genes modulate the neuronal apoptosis following ischemia brain damage.
目的探讨三七三醇皂苷对局灶性脑缺血损伤的保护作用。
Objective To study the protective effects of PTS on focal cerebral ischemia in rats.
而星形胶质细胞的激活支持和保护大脑免受脑缺血损伤。
The activation of astrocytes help ischemic brain repair and prevent from injury.
目的：探讨麝香酮减轻兴奋毒性、抗急性脑缺血损伤的作用。
Objective: To study the effect of muscone on attenuating excitotoxity and brain damage in acute cerebral ischemia.
表明HLJDTAF对局灶性脑缺血损伤有明显的治疗作用。
Protective effect of HLJDTAF on experimental cerebral Ischemia In rat.
方法采用线栓法建立雌性大鼠永久性局灶性脑缺血损伤模型。
METHODS The model of permanent focal cerebral ischemia was established with Zea-Longa occluding suture in female Sprague-Dewley rats.
目的：探讨微栓子不同组成和大小与脑缺血损伤程度的相关性。
Aim: To study the correlation between the cerebral ischemic injury and the microemboli of different composition and size.
目的探讨IL-10对大鼠局灶性脑缺血损伤的保护作用。
OBJECTIVE To study the protective effect of IL - 10 on the injury by focal cerebral ischemia in rats.
目的：观察氯丙米嗪对局灶性脑缺血损伤后大鼠运动功能的作用。
OBJECTIVE: To observe the effect of chlorimipramine on motor function of rats with focal cerebral ischemic injury.
背景如何减轻脑缺血损伤仍是一个世界性的难题和亟待解决的问题。
Background How to reduce cerebral ischemic injury remain a thorny problem, also an urgent and unresolved issue.
结论：EAAC 1反义寡核苷酸对急性脑缺血损伤有神经保护作用。
CONCLUSION: EAAC1 antisense oligonucleotide has the neuroprotective effect against acute cerebral ischemia injury.
结论：急性局灶性脑缺血损伤中有白细胞流变学异常，变形能力下降。
CONCLUSION: the acute focal cerebral ischemic injury leads to the abnormal leukocyte rheology and declining of deformability.
结扎并低氧处理7日龄大鼠右侧颈总动脉制备半球性脑缺血损伤模型。
The animal models of cerebral hypoxia-ischemia were prepared in 7 days old rats by systemic hypoxia for 2h and right common carotid artery ligation.
结论：参附注射液对大鼠短暂性局灶性脑缺血损伤呈非剂量依赖性保护作用。
Conclusions:The Shenfu injection possesses dosageindependent protective effects on transient focal cerebral ischemic injury.
目的：观察补阳还五汤对大鼠脑缺血损伤后感觉运动功能和脑损伤的长期影响。
AIM To observe the long-term cerebroprotective effects of Buyang Huanwu decoction BYHWD on the sensorimotor function and brain damage after focal cerebral ischemia in rats.
因此细胞因子及其拮抗剂的研究有可能为脑缺血损伤的治疗提供一种新的思路和手段。
Study on the cytokines and their antagonists provides a new method for the therapy of cerebral ischemic injury in the future.
它选择性地作用于内皮细胞、促进新生血管形成；它参与脑缺血损伤的病理生理过程。
It ACTS selectively on endothelial cells to promote angiogenesis and takes part in pathophysiology process of cerebral ischemia injury.
体外细胞培养及动物实验证明，促红细胞生成素对脑缺血损伤具有神经保护和治疗作用。
It has been proved that erythropoietin has neuroprotective effect and therapeutic action on cerebral ischemia in vivo and in vitro.
目的：探讨不同时间、不同剂量硫酸镁对大鼠永久性局灶性脑缺血损伤后神经功能的保护作用。
AIM: To investigate the neuroprotective effects of MgSO_4 in different doses at varying stages after permanent focal cerebral ischemia.
结论：HGF在局灶脑缺血诱导的高表达可能在脑缺血损伤组织的保护及修复过程中起重要作用。
Conclusion: the high expressions of HGF in rat brain might play an important role in the protection and repair of the damaged tissue induced by focal cerebral ischemia.
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral ischemic injury, etc. were reviewed and...
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral...
目的：评价大黄素抗脑缺血损伤作用，并从细胞因子水平及表达方面探讨其抑制炎性级联反应机制。
Objective: to assess emodin antagonism to cerebral ischemia injury, and to discuss the mechanism of emodin inhibiting the inflammatory cascade reaction from the levels and expressions of cytokines.
结论：降低MCAT大鼠的全血黏度，抑制血栓形成，可能是EGB抑制脑缺血损伤的作用机制之一。
Conclusion: Decreasing blood viscosity in MCAT rats and inhibiting thrombosis are probably the mechanism of EGB anti-ischemic damage.
越来越多的研究证实，凋亡在轻、中度暂时性脑缺血损伤所致的神经细胞迟发性死亡过程中起重要作用。
A number of studies have provided evidence that apoptosis might be an important factor in the progression of tissue damage resulting from cerebral ischemia.
目的研究聪圣胶囊对小鼠脑缺血再灌后脑组织自由基变化的影响，分析聪圣胶囊抗脑缺血损伤的作用机制。
Object To investigate the effects of Congsheng Capsule (CSC) on free radical change after cerebral ischemia reperfusion, and analyze the mechanisms of CSC anti cerebral ischemia action.
结论：IL -8具有双重作用，既参与脑组织的正常代谢及生理功能，又参与了脑缺血损伤的病理过程。
Conclusion: IL 8 has double roles: it plays a role in metabolism and function in normal brain tissue and a role in pathological course of cerebral ischemia injury.
结论：局灶性脑缺血损伤可诱导神经元egfr表达增加，EGFR表达增加可能与缺血神经元自身保护作用有关。
CONCLUSION: the increase of neuron EGFR expression can be induced by focal cerebral ischemia injury, and may be related to the self protection of ischemia neurons.
结论：局灶性脑缺血损伤可诱导神经元egfr表达增加，EGFR表达增加可能与缺血神经元自身保护作用有关。
CONCLUSION: the increase of neuron EGFR expression can be induced by focal cerebral ischemia injury, and may be related to the self protection of ischemia neurons.
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