分析该病可能是一种线粒体肌病，属脂质沉积性肌病。
We thought that the "white-body" disease should be one of mitochondrial myopathy, was lipid storage myopathy.
促进氧化低密度脂蛋白的形成，促进脂质沉积于动脉壁等。
It may lead to increase susceptibility of low density lipoprotein to oxidation that makes them more atherogenic.
脂质沉积性肌病是一种肌细胞内脂肪异常沉积引起的常染色体隐性遗传病。
Lipid storage myopathy is one kind of autosomal recessive inherited disease with lipid abnormally sludging in the muscle cell.
结论：睾酮可能通过对血浆脂质代谢的影响促进高脂血症大鼠主动脉脂质沉积。
Conclusions: Testosterone increase aortic lipid deposition in the hypercholesterolemic rats by means of the influence on plasma lipid metabolism.
结果高脂组心肌组织中有粥样硬化斑块的形成，间质内有大量脂质沉积、浸润。
Results Atherosclerotic plaques and extensive lipid infiltration appeared in myocardium tissue and cell matrix of hyperlipidemia animals separately.
结果部分照射后存活的细胞有超微结构的改变：线粒体肿胀、脂质沉积，粗面内质网扩张。
Results Changes in ultrastructure observed in part of the alive cells were as following: swollen mitochondria, lipid deposit, rough endoplasmic reticulum dilatation.
目的分析3例被误诊的脂质沉积性肌病的临床及病理特点，探讨脂质沉积性肌病被误诊的原因。
Objective to study the clinical and pathological features of 3 cases of misdiagnosised lipid storage myopathy (LSM), and further discover the reasons of diagnosis error in this disease.
据专家解释，这种现象非常罕见，原因是脂蛋白代谢异常或脂质沉积症，目前尚不清楚血液变色原因。
According to experts, this phenomenon caused bymetabolic abnormality of lipoprotein or lipoidosis is very rare. The reasonof the changing of the blood color is still unknown.
电镜观察显示野生马齿苋能有效地减轻主动脉壁脂质沉积，并有抑制动脉粥样硬化斑块形成的作用。
It was proved under the observation of electron microscope that purslane could lower lipids deposition of aorta intima and inhibit the formation of atherosclerotic plaque.
单纯移植组的颈静脉移植段仅有明显的内膜增生和平滑肌细胞表型转换及增殖，未见明显脂质沉积；
But only intimal hyperplasia and the migration of smooth muscle cells were observed in the vein graft group.
结论高脂喂养可增加糖尿病大鼠血浆脂质水平，引起肌肉组织内脂质沉积，导致胰岛素抵抗(IR)。
Conclusion High fat diet increase the level of plasma fatty, result in lipid deposition in muscle and insulin resistance (IR).
结论：金力源胶囊可减少高脂血症大鼠肝脏脂质沉积，降低肝脏脂质过氧化水平，恢复肝脏抗氧化能力，预防脂肪肝的形成。
Conclusion: JLY could reduce liver lipid deposition and lipid peroxidation, restore liver antioxidant activity in rats with hyperlipidemia, which could prevent hepatocyte from fatty degeneration.
方法择取脂质沉积性肌病伴血清肌酶明显升高的病人14例，将临床表现、血清肌酶、肌电图和肌肉病理及肌肉组织化学改变进行总结分析。
Methods 14 cases of LSM with high level of CK in serum were analyzed clinically and compared with their muscle morphological and histochemical changes.
细胞损伤时细胞内的沉积物是多种多样的。肝细胞损伤尤其是长期酒精性损伤导致脂蛋白大量异常转运从而使脂质在肝实质细胞内大量沉积。
The liver injury with chronic alcoholism leads to fibrosis and regeneration of the hepatocytes in nodules. This firm, nodular appearance of the liver as seen here is called cirrhosis.
目的探讨神经元腊样质脂褐素沉积病（NCL）的临床表现规律和影像学特点。
Objective To study the clinical and neuroimaging features of neuronal ceroid lipofuscinosis (NCL) in China.
近年来研究认为SAA是一种新的载脂蛋白，发现SAA能通过抑制HDL对胆固醇逆转运过程，增加脂质在AS病灶的沉积。
SAA is a kind of apoprotein, it can inhibiting HDL's RCT function, and through this way increase serum cholesterol level and increase cholesterol deposit to as site.
近年来研究认为SAA是一种新的载脂蛋白，发现SAA能通过抑制HDL对胆固醇逆转运过程，增加脂质在AS病灶的沉积。
SAA is a kind of apoprotein, it can inhibiting HDL's RCT function, and through this way increase serum cholesterol level and increase cholesterol deposit to as site.
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