其次，是脂毒性问题。
目的探讨针刺是否具有抑制脂毒性的作用，及其可能机制。
Objective to investigate if acupuncture has an inhibitory effect on fatty toxicity and its possible mechanism.
心脏中脂质大量积聚，可以引起心脏功能异常，称之为脂毒性心肌病。
Lipid accumulation in the heart may lead to cardiac dysfunction, which is called lipotoxic cardiomyopathy.
心肌游离脂肪酸的摄入和利用的失协调导致心肌中脂质积聚并进而引起脂毒性心肌病。
Mismatches between myocardial fatty acid uptake and utilization lead to the accumulation of cardiotoxic lipid species, and cause lipotoxic cardiomyopathy.
结论针刺可以降低肥胖患者的游离脂肪酸水平，提高其胰岛素敏感性，从而表现出抑制脂毒性的效应。
Conclusion Acupuncture can lower the levels of free fatty acids in the patients and increase their sensitivity to insulin to inhibit the effect of fatty toxicity.
研究过氧化物酶体增殖剂活化受体在脂毒性心肌病中的作用，可以为预防和治疗脂毒性心肌病提供新思路。
Studies of the role of the peroxisome proliferators-activated receptor plays in lipotoxic cardiomyopathy will provide new strategies to prevent and cure lipotoxic cardiomyopathy.
将脂质吸收到脂肪组织是人体处理这种毒性的一种途径。
Absorbing them into adipose tissue is one of the body's ways of dealing with that toxicity.
脂多糖还能绑定到血浆白蛋白形成复合物，增加LPS的毒性以及LPS对膜受体的亲和力。
LPS can bind to plasma proteins and form complexes that increase toxic activity of LPS and affinity of LPS to cell receptors.
提示脂质过氧化可能是砷毒性作用的机制之一。
It indicates that lipid peroxidation may be one of the mechanisms of liver and brain injury in arsenic poisoning.
结论氟有潜在的致畸性和胚胎毒性，胚胎组织gsh活性和卵黄囊细胞膜脂流动性降低可能在氟致胚胎发育毒性中起重要作用。
Conclusions Fluoride could result in embryonic growth retardation and potential teratogenic toxicity. These effects might be due in part to decrease in GSH activity and membrane lipid fluidity.
润滑油、脂的毒性主要来源于添加剂和填充物，其主要危害是皮炎。
The toxicity of lubricating oil and grease 'chiefly stem from additives and stuff. Its main harm is dermatitis.
苯妥英钠对缺血性脑损伤的神经保护作用与其能抑制自由基形成，减轻自由基介导的毒性，抗脂质过氧化反应以及扩张血管，增加局部脑血流量有关。
Phenytoin sodium may protect brain tissues from cerebral ischemic injury by inhibiting formation of free radical, anti-lipid peroxidation and improving regional cerebral blood flow.
结论微囊藻毒素- LR在非细胞毒性的剂量下可引起肝细胞的氧化应激，但尚不足以导致脂质过氧化。
Conclusion The dose of MCLR that is not cytotoxic can induce hepatocyte oxidative stress, which can not be identified as lipid peroxidation yet.
目的探讨氯化镉对NRK大鼠肾细胞毒性作用和脂质过氧化的影响。
Objective to investigate the effects of cadmium chloride on cytotoxicity and lipid - peroxidation reaction in NRK cells.
目的研究排毒清脂胶囊对大鼠的长期毒性。
Objective to study the long-term toxicity of Paidu Qingzhi capsule in rats.
结论2 -乙氧基乙醇能引起肝脏一氧化氮自由基增加并诱发脂质过氧化反应对肝脏发生毒性作用。
Conclusion the study demonstrated that 2 ethoxyethanol can increase the liver level of no free radicals and the lipid peroxidation may play an important role in 2 ethoxyethanol's liver toxicity.
目的：研究p选择素在高胆固醇血症小鼠肾脏的表达，探讨脂质肾毒性损伤的机制。
Objective: to detect the expression of P-selectin in mice with hypercholesterolemia, and to explore the mechanism of toxic injury to the kidney induced by lipid.
目的：研究氡及其子体和苯对小鼠血液和脂质过氧化以及骨髓毒性的联合作用及其类型，为进一步探讨二者联合毒作用的机理提供实验依据。
Objective: To study the joint effects of radon and benzene on the blood and bone marrow of mice to provide experimental data for further study.
阳离子脂质体的毒性主要来自于小分子阳离子脂质分子。
Cytotoxicity of cationic liposome is mainly engendered by cationic lipid.
脂质过氧化反应产生的终产物mda具有细胞毒性，所以可以通过测mda的含量来评价细胞质膜的受损程度。
Produced by lipid peroxidation end products MDA cytotoxicity, so the content of MDA can be measured to assess the extent of the damage of cell plasma membrane.
通过在药物组合物及包含LL - 37的生长培养基中包括成双层性极性脂质，具体为双半乳糖基甘油二酯，可降低LL - 37的细胞毒性作用。
The cytotoxic effect of LL-37 may be reduced by including a bilayer-forming polar lipid, especially a digalactosyldiacylglycerol, in pharmaceutical compositions and growth media comprising LL-37.
嗜热菌杀酵母素通过耗尽细胞的鞘脂类诱导凋亡，抑制白介素2依赖性的小鼠毒性细胞的增殖，是一种有效的免疫抑制剂。
Myriocin induces apoptosis by depletion of cellular sphingolipids, inhibits proliferation of IL-2-dependent mouse cytotoxic cells and is a potent immunosuppressant.
嗜热菌杀酵母素通过耗尽细胞的鞘脂类诱导凋亡，抑制白介素2依赖性的小鼠毒性细胞的增殖，是一种有效的免疫抑制剂。
Myriocin induces apoptosis by depletion of cellular sphingolipids, inhibits proliferation of IL-2-dependent mouse cytotoxic cells and is a potent immunosuppressant.
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