慢性接触镉主要引起肾小管损伤，表现为蛋白尿、糖尿和氨基酸尿。
Chronic exposure to Cd mainly causes the renal proximal tubule damage, which represents that the excretion of proteinuria, glucosuria and aminoaciduria.
如果不进行治疗，肌红蛋白尿就可能造成肾小管坏死。
Myoglobinuria may result in renal tubular necrosis if untreated.
药物从肾脏排泄可损害肾小管，产生蛋白尿、血尿，血中尿素氮、肌酐升高等肾功能损害的表现。
Drug can be excreted from the kidney tubular damage, resulting in proteinuria, hematuria, blood urea nitrogen, creatinine, and other higher Renal damage.
但是循环到血液中会被肾屏蔽，积聚起来的血红蛋白阻塞了肾小管，严重时对肾脏造成永久性伤害。
Once released into the blood, it is filtered by the kidney, where it accumulates, blocks microscopic tubules and damages the organ, sometimes permanently.
慢性接触镉主要引起肾小管损伤，表现为蛋白尿、糖尿、氨基酸尿、酶尿及尿镉排出量明显增加等。
Chronic exposure to Cd mainly causes the renal proximal tubule damage, which represents that the excretion of proteinuria, glucosuria, aminoaciduria, enzymaticuria and urinary cadmium increase.
而高血压早期肾脏就出现了以尿微量蛋白等为特征的肾小管肾小球的功能异常。
And the disfunctions of nephric tubule and glomerulus characterized by renal small protein have appeared in the earlier period of the hypertensive kidney.
背景：钠-葡萄糖协同转运蛋白2（SGLT2）在近端肾小管表达，负责重吸收肾小管液中90%的葡萄糖。
Context: Sodium-glucose cotransporter 2 (SGLT2) expressed in the proximal renal tubules accounts for about 90% of the reabsorption of glucose from tubular fluid.
引起肾小管间质病变的原因比较复杂，其中蛋白尿的肾损伤作用已为大量临床和实验研究所证实。
The causes for tubulointerstitial changes remain rather complex and the role of proteinuria in damaging nephridial tissue has been demonstrated by large number of clinical and experimental studies.
共聚焦显微镜分析显示正常asbt蛋白主要定位于肾小管上皮细胞膜上，与生物信息学的预测结果一致。
Confocal scanning analysis showed that ASBT protein was mainly localized at the cellular plasma membrane, consistent with the predicting result obtained by bioinformatics.
结果根据尿蛋白分子量大小可以将其区分为肾小球性、肾小管性、混合性、溢出性及生理性蛋白尿。
Results We can discriminate glomerular proteinuria, tubuloproteinuria, mixed albuminuria, overflow proteinuria and physiologic proteinuria according to molecular weight of urine protein.
探讨白蛋白对近端肾小管上皮细胞增殖与凋亡的影响。
To study the effects of albumin overload on proliferation and apoptosis in proximal tubular epithelial cells.
目的探讨灯盏花素对糖尿病大鼠肾小管-间质细胞间黏附因子- 1 (ICAM - 1)与单核细胞趋化蛋白- 1 (MCP - 1)表达的影响。
Objective To study the effect of breviscapine on intercellular adhesion molecule-1 (ICAM-1) and monocyte chemotactic protein-1 (MCP-1) expression in tubulointerstitium of experimental diabetic rats.
其中肾小球性蛋白尿13例、肾小管性蛋白尿9例、混合性蛋白尿11例、溢出性蛋白尿2例、生理性蛋白尿5例。
Cuj. Cujus 13 of them were glomerular proteinuria, 9 tubuloproteinuria, 11 mixed albuminuria, 2 overflow proteinuria and 5 physiologic proteinuria.
由于转运蛋白的特异性之间存在重叠，药物在肾小管分泌过程中产生的药物的相互作用将是今后临床上值得注意的问题。
Due to overlapping specificities of the transport proteins, drug interactions at the level of tubular secretion is an event that may occur in clinical situation.
免疫组化染色表明ASBT蛋白主要表达于人近端肾小管上皮的刷状缘侧，在间质及远端小管没有表达。
The results of immunohistochemistry revealed that ASBT was expressed at brush border membrane of proximal renal tubular cells, but not expressed in distal tubule and renal interstitium.
现通过介绍几种以低分子质量蛋白尿为主要特点的遗传性肾小管疾病，以提高对此类疾病的认识，避免不必要的治疗。
Herein several inherited renal tubular diseases manifested of low molecular weight proteinuria were introduced in order to improve the understanding of these diseases and avoid unnecessary treatment.
现通过介绍几种以低分子质量蛋白尿为主要特点的遗传性肾小管疾病，以提高对此类疾病的认识，避免不必要的治疗。
Herein several inherited renal tubular diseases manifested of low molecular weight proteinuria were introduced in order to improve the understanding of these diseases and avoid unnecessary treatment.
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