结论STAT 3蛋白表达增强可能介导缺血性脑损伤的信号转导过程，其在脑缺血损伤神经元凋亡过程中起关键作用。
Conclusion the expressions of STAT3 increased might indicate the transmission course in ischemic nerve cell signal, and play a key role in neuron apoptosis of ischemic brain injury.
电针可以增加大鼠脑缺血区皮层ngf的分泌和表达时程，这可能是其保护缺血性脑损伤的机制之一。
Ea can increase the secretion and expression period of NGF in rats ischemic cortex, and this may be one of its protective mechanisms for ischemic cerebral damage.
目的：研究慢性脑缺血时大鼠大脑皮质中神经生长因子(NGF)的表达变化，探讨缺血性脑损伤及修复机制。
Objective: to investigate the expression of nerve growth factor (NGF) in cerebral cortex of chronic cerebral ischemia rats, and to explore the mechanism of cerebral ischemia damage and recovery.
目的：研究慢性脑缺血时大鼠大脑皮质中神经生长因子(NGF)的表达变化，探讨缺血性脑损伤及修复机制。
Objective: to investigate the expression of nerve growth factor (NGF) in cerebral cortex of chronic cerebral ischemia rats, and to explore the mechanism of cerebral ischemia damage and recovery.
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