分别在缺氧前给予精胺，缺氧-复氧后给予精胺、精脒、腐胺。
Spermine was added extracellularly to the bath before anoxia and spermine, spermidine or putrescine was added extracellularly after reoxygenation.
目的：观察外源性低浓度多胺对大鼠缺氧-复氧心肌细胞钙超载的影响。
AIM: To study the effect of extrogenous low concentration polyamine on cardiomyocyte calcium overload caused by anoxia and reoxygenation.
方法：运用全细胞膜片钳技术记录持续性钠电流，并观察其在缺氧-复氧模型下的变化。
Methods: the whole cell patch clamp technology was used to record this current and study its change under the condition of hypoxia-reoxygenation model.
目的明确异氟醚减少离体鼠肝缺氧-复氧损伤的功能主要发生在哪一阶段，以及和枯否细胞的关系。
Objective To investigate which stage is the most important and the relationship with kupffer cell, during isoflurane decrease anoxia-reoxygenation injury in isolated rat liver.
摘要目的明确异氟醚减少离体鼠肝缺氧-复氧损伤的功能主要发生在哪一阶段，以及和枯否细胞的关系。
Abstract Objective To investigate which stage is the most important and the relationship with kupffer cell, during isoflurane decrease anoxia-reoxygenation injury in isolated rat liver.
细胞形态呈现多样化，如圆形、梭形及椎形。缺氧复氧损伤后心肌细胞伪足缩短或消失，折光性下降，搏动减弱或消失。
ResultsBeing injured by hypoxia and reoxygenation, refracting power of the cells declined, pseudo-podium became shortened or disappeared, cell beat became weak or stopped.
海马神经元；缺氧复氧损伤；脑神康胶囊；抗氧化；凋亡。
Hippocampal neurons; Anoxiareoxygenation; Naoshenkang capsule; oxygen free radical; Apoptosis.
脑微血管内皮细胞缺氧复给氧损伤后，采用流式细胞仪检测细胞凋亡的程度。
Apoptosis was detected with flow cytometry after hypoxia-reperfusion oxygen damage of the cerebral microvascular endothelial cells.
探讨缺氧复给氧损伤后脑微血管内皮细胞凋亡的机制。
To explore the mechanism of apoptosis after hypoxia-reperfusion oxygen damage the cerebral microvascular endothelial cells.
结论七氟醚预处理抗缺氧复氧后大鼠海马神经元凋亡的作用呈剂量依赖性。
Conclusions Sevoflurane 6% inhaled preconditioning can obviously inhibit apoptosis of hippocampal neurons by hypoxia - reoxygenation in rats.
结论七氟醚预处理抗缺氧复氧后大鼠海马神经元凋亡的作用呈剂量依赖性。
Conclusions Sevoflurane 6% inhaled preconditioning can obviously inhibit apoptosis of hippocampal neurons by hypoxia - reoxygenation in rats.
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