目的探讨急性心肌缺血不同时间心肌缝隙连接蛋白43的分布特征。
Objective To characterize the distribution pattern of gap junctional connexin43 during different time session in rabbit with early myocardial ischemia.
贝那普利及缬沙坦可有效减轻心房纤维化和心肌缝隙连接蛋白的重构。
Benazepril and valsartan can decreased the degree of atrial fibrosis and remodeling of Gap junction connexin effectively.
前言缝隙连接是由连接蛋白组成的细胞膜通道，其属于多基因家族。
PrefaceGap junctions are cell membrane protein channels composed of connexin proteins, which belong to multigene family.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43的表达受到抑制可能是导致心肌细胞缝隙连接通讯障碍以致心律失常的一个原因。
Conclusion in viral myocarditis, the expression of connexin 43 in the involved cardiac muscle cells was restrained, that may result in dysfunction of gap junction communication.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43和结蛋白的表达受到抑制，可能是导致心肌细胞缝隙连接通讯和收缩功能障碍以致心律失常的一个原因。
Conclusion in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43和结蛋白的表达受到抑制，可能是导致心肌细胞缝隙连接通讯和收缩功能障碍以致心律失常的一个原因。
Conclusion in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
应用推荐