细胞内线粒体凋亡途径是凋亡的主要途径之一。
The participation of mitochondria in apoptosis is one of the most important pathways.
表面上看，线粒体的重新激活同样会促进凋亡发生。
It seems this reactivation also allows the mitochondria to stimulate apoptosis.
线粒体相当于细胞的能源植物，当自由基干涉其生物过程时，线粒体会接受信号开始细胞凋亡程序。
Mitochondria act as cellular energy plants, and when free radicals interfere with their biochemical pathways, mitochondria receive a signal to start cell death.
依赖于半胱氨酸蛋白酶的激活，美沙酮通过激活线粒体克服了白血病细胞中的阿霉素耐药、多药耐药、和凋亡抑制。
Depending on caspase activation, methadone overcomes doxorubicin resistance, multidrug resistance, and apoptosis resistance in leukemia cells through activation of mitochondria.
线粒体在能量代谢、自由基产生、衰老、细胞凋亡中起重要作用。
Mitochondria play an important role in energy metabolism, free radical production, aging and cell apoptosis.
目的观察线粒体功能失调在缺血再灌后神经元凋亡中的作用。
Objective to observe the role of mitochondria dysfunction in neuron apoptosis during ischemia-reperfusion injury.
结论EGCG通过活化线粒体信号传导途径诱导BGC823细胞凋亡。
Conclusion EGCG induces apoptosis of BGC823 cells via activating mitochondrial signal transduction pathway.
线粒体是哺乳动物的产能、供能细胞器，与生长、发育、衰老和凋亡等多种细胞事件及疾病有关。
Mitochondria, which can produce and supply energy for mammals, are involved in many cellular events of growth, development, aging, apoptosis as well as diseases.
结论NO介导的小鼠胸腺细胞的凋亡过程，依次为磷脂酰丝氨酸外翻、线粒体去极化、CL氧化及细胞皱缩。
CONCLUSION: The process of thymocyte apoptosis mediated by NO in mice is successively PS ectropion, mitochondrial depolarization, CL oxidation and cell crenation.
D半乳糖；摘除胸腺；神经细胞凋亡；线粒体膜电位；衰老模型。
Dgalactose; Thymus gland removal; Nerve cells apoptosis; Mitochondria membrane potential; Aging model.
它发挥保护作用的可能机制，主要通过异丙酚对氧自由基、钙离子的调节、细胞因子、细胞凋亡和线粒体等几方面的作用来实现。
The propofol play its role through many possible mechanism such as the modulation of oxygen free radical and calcium, cell apoptosis, and mitochondrion.
线粒体在脑缺血神经元凋亡的发生过程中起着关键作用。
Mitochondra play a key role in the process of cerebral ischemic neuronal apoptosis.
结论铝可以诱导大鼠海马神经细胞凋亡，并引起线粒体结构和线粒体酶活力的改变。
Conclusion al can induce apoptosis of neurocyte in rats' hippocampus, and it can result in changes of the structure of mitochondria and mitochondrial enzymes.
目的探讨白藜芦醇诱导鼻咽癌细胞cne2z凋亡的线粒体机制。
AIM to study whether resveratrol induced apoptosis in nasopharyngeal carcinoma cells CNE 2z is via a mitochondrial pathway.
线粒体不仅是细胞能量产生的重要场所，而且参与了细胞凋亡、死亡的过程。
Mitochondria are not only an important place generating cellular energy, also involved in the apoptotic and death process of cells.
线粒体与内质网是细胞凋亡信号传导途径中起重要作用的细胞器。
Mitochondria and endoplasmic reticulum are vital cell organs in the process of transmitting apoptosis signs.
ROS通过氧化线粒体心磷脂、线粒体dna和线粒体重要的蛋白质对线粒体造成氧化损伤，进而诱导细胞凋亡。
Through oxidizing the mitochondrial cardiolipin, mitochondrial DNA and important proteins in mitochondria, ROS leads to the oxidative stress towards mitochondria, further induces the cell apoptosis.
脑缺氧后神经元线粒体损伤不单使细胞发生能量缺失和功能丧失，还可以介导凋亡调节信号，是缺氧损伤后神经元凋亡的一个中心环节。
Impairment of neuronal mitochondria following hypoxia of brain not only result in nerve cell's energy-deprivation and dysfunction, mitochondria also play key roles in apoptosis of neurons.
检测转染组和未转染组顺铂杀伤率、细胞凋亡和线粒体膜电位的变化。
Using MTT and flow cytometer, we detected the changes of cell mortality rate, apoptosis and mitochondrial membrane potential.
目的研究糖尿病大鼠小肠平滑肌细胞线粒体膜电位变化与细胞凋亡的关系。
Objective To investigate the relationship between mitochondrial membrane potential change and apoptosis of smooth muscle cells of the small intestine in diabetic rats.
结论镉诱导HEK293细胞的凋亡可能通过线粒体途径。
CONCLUSION Cd-induced apoptosis involves mitochondrial pathway in HEK293 cells.
结论过钒酸钠可逆转电离辐射诱导的细胞线粒体膜电位的降低，减少造血细胞凋亡的发生。
Conclusion Per reversed the repression of mitochondrial membrane potential and suppress apoptosis induced by ionizing irradiation in the hematopoietic cells.
结果心肌缺血-再灌注时能量耗竭、自由基形成、钙超载、细胞凋亡等过程中线粒体发挥重要的作用。
Results the mitochondrion plays an crucial role in the processes of energy burn-out, free radical formation, calcium overload and cells apoptosis during myocardial ischemia-reperfusion.
线粒体是真核生物能量和代谢的中心，也是在细胞凋亡、信号转导中起关键调节作用的细胞器。
Cytomicrosome is the center of energy and metabolism of eukaryon. It is also a cellular organ with the critical adjusting function in processes of apoptosis signal.
目的探讨表没食子儿茶素没食子酸酯(EGCG)通过活化线粒体途径诱导人胃癌BGC823细胞凋亡的作用机制。
Objective to elucidate the mechanism of apoptosis induced by epigallocatechin-3-gallate (EGCG) on human gastric cancer BGC823 cells via mitochondrial pathway.
结论：苦参碱能诱导C6胶质瘤细胞的凋亡，其机制与死亡受体途径和线粒体途径的众多基因有关。
Conclusion: Matrine can induce C6 glioma cell apoptosis and the mechanism might be related to a number of genes involved in death receptor pathway and mitochondrial pathway.
目的研究地塞米松(DEX)诱导小鼠胸腺细胞凋亡进程中线粒体去极化作用特点。
Objective to study on the dexamethasone (dex) -induced mitochondrial depolarization and apoptosis process in mouse thymocyte.
这和最近的研究是一致的，该研究表明卵母细胞成熟抑制因子能诱导用TNF处理的人类神经多肽的凋亡而不从线粒体中释放出来（7）。
This is in accord with a recent study that shows Omi can induce apoptosis in human neutrophils treated with TNF- without being released from the mitochondria (7).
在细胞凋亡的过程中，线粒体起着主要的作用。
Mitochondria play a crucial role in cellular survival and apoptosis.
近年来对凋亡发生和调控机制的研究表明，细胞凋亡途径主要有两条：线粒体途径和死亡受体途径。
In recent years, studies of occurrence and regulation mechanism of apoptosis show that the two major apoptotic pathways are the mitochondrial and death receptor pathways.
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