以流式细胞术检测各组窦房结细胞存活率；
方法运用穿孔膜片箝技术，在游离的兔心窦房结细胞上进行研究。
MethodsPerforated patch clamp techniques were employed on isolated rabbit SN cells.
现就窦房结细胞起搏基因hcn4的特性及其与窦房结功能之间的关系作进一步研究和探讨。
This article mainly reviews the features of SAN pace-making gene HCN4 in relation to SAN function.
结论用甲醛湿敷法损伤窦房结组织不仅导致窦房结细胞不可逆性损伤，而且可诱发窦房结细胞凋亡。
Conclusion Formaldehyde wet dressing method can not only lead to irreversible damage of the SAN cells but evoke the apoptosis of SAN cells as well.
正常情况下，窦房结产生规律的电脉冲，传入各心房，再带动细胞同步收缩。
Normally the sinoatrial node generates regular electrical pulses that spread across the Chambers of the heart, causing the cells to contract in synchrony.
窦房结的心肌细胞：这张图显示了窦房结的切面，右心房的一块起搏区。
Cardiomyocytes in the sinoatrial node This image shows a section of the sinoatrial node, a pacemaking area of the right atrium of the heart.
细胞的形态和结构介于P细胞和心肌细胞之间。 最后对窦房结内各细胞的功能特点加以讨论。
The shape and structure of T cell is between P and myocardial cell, and situated on the periphery of node.
结果：窦房结和房室结内的细胞主要为P细胞和T细胞。
Results: The SAN and AVN chiefly consisted of P cells and t cells.
窦房结和房室结周围神经节中的部分神经细胞也呈免疫阳性反应。
Some neural cells in ganglions surrounding sino atrial node and atrio ventricular node were also positive to neurofilament.
窦房结中心区的细胞集落，全都存在于窦房结动脉周围。
These cell clusters in the central part of the sinus node are all around the sinus node artery.
鉴于该病常伴随缺血性心脏病而出现，故窦房结缺血再灌注诱导细胞凋亡可能为其原因之一。
Because it is often accompanyed by ischemic heart diseases, apoptosis resulting from ischemia-reperfusion of sinus node maybe the one of the reasons.
光镜和透射电镜观察窦房结组织及其细胞、间质的结构变化。
Histologic changes of SAN were studied through serial sections and transmission electron.
光镜和透射电镜观察窦房结组织及其细胞、间质的结构变化。
Histologic changes of SAN were studied through serial sections and transmission electron.
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