突触可塑性常常影响突触结构的形状。
Synaptic plasticity often affects the shape of synaptic structures.
记忆的保持——突触稳定性还是突触可塑性?。
Memory retention-the synaptic stability versus plasticity dilemma Wickliffe C.
短时程突触可塑性包括短时程增强和短时程压抑两种类型。
Short - term synaptic plasticity includes two forms, short - term enhancement and short - term depression.
特别是丙戊酸钠对突触可塑性影响的报道很少，结果也不一致。
So far, there were few studies about effects of VPA on synaptic transmission and the data presented were not consistent.
该中心旨在通过跨学科的协作方式，提高人类对神经突触可塑性的了解。
The centre's mission is to advance our understanding of synaptic plasticity in a multidisciplinary and collaborative manner.
突触的数量和功能发生改变可引起突触可塑性的改变，进而影响学习记忆能力。
Changes in the number and function of synapsis can cause changes in synaptic plasticity, thereby can affect learning and memory.
突触可塑性是神经系统所具有的重要特征，也是神经系统实现其功能的重要保障。
Synaptic plasticity is an important characteristic of neural system. It is significant to the function of neural system.
突触后致密区与突触可塑性的关系十分密切，对于维持突触发挥正常功能至关重要。
Postsynaptic dense area and the close relationship between synaptic plasticity are essential for the maintenance of synaptic function properly.
已有的电生理学实验已经表明，褪黑素可以调节海马神经元的兴奋性和突触可塑性。
Previous electrophysiological experiments have revealed that melatonin modulates hippocampal neuronal excitability and plasticity.
突触可塑性对于脑发育过程中的神经环路重构以及学习记忆等脑的高级功能是非常重要的。
Synaptic plasticity is important for the remodeling of neural circuits during brain development and is implicated in higher brain functions such as learning and memory.
已有广泛的研究表明突触可塑性如长时程增强和长时程抑制可以作为学习和记忆机制的细胞模型。
Extensive studies indicated that synaptic plasticity, such as long-term potentiation or long-term depression, is the cellular basis for learning and memory.
并且在不同发育期其对突触可塑性的损害不同，其中哺乳期是大鼠对PBDE209最敏感的时期。
We studied the effects of PBDE209 on synaptic plasticity in the DG in vivo, and discussed different effects of PBDE209 in the different developmental periods.
大量实验数据证明，NR2B亚单位与哺乳动物的突触可塑性、学习记忆、痛觉和神经系统疾病有关。
Accumulating evidence indicates that, NR2B is crucial for synaptic plasticity, learning and memory and nervous system degenerative diseases.
海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
结论环境刺激对HIBD大鼠功能的促进作用可能与突触超微结构参数的改变，增强突触可塑性有关。
Conclusion The role of environmental stimulation in functional outcome of HIBD is likely to be related to change of synaptic substructure and increased synaptic plasticity.
在中枢神经系统，CREB调节着神经细胞生长发育，参与神经细胞突触可塑性、长时程记忆的形成过程。
In central nerve system, CREB participates in promoting neuron growth, the processes of plasticity and long term memory.
当开始了解这些突触改变的细胞生物学机制的时候，也应该考虑这种突触可塑性在完整大脑中的功能意义。
Yet, while we are beginning to understand the cellular mechanisms underlying synaptic changes, it is important to consider the functional implications of synaptic plasticity in the intact brain.
本课程探讨神经元传导，可激发细胞膜上之离子通道，神经突触传导与神经突触可塑性的分子以及细胞机制。
Surveys the molecular and cellular mechanisms of neuronal communication. Coversion channels in excitable membrane, synaptic transmission, and synaptic plasticity.
目的观察大鼠脑梗死及电针干预后突触素(syp)的动态变化，探讨突触可塑性的物质基础、机制及针刺的影响。
Objective To explore the mechanism, material fundamental of synapse plasticity and acupuncture's efficacy by watching the alteration of SYP after cerebral infarction and acupuncture treatment.
腹侧被盖区多巴胺神经元的突触可塑性的改变可能在许多药物滥用(包括可卡因滥用)所导致的回路修饰中起着一定作用。
Changes in synaptic plasticity in dopamine neurons of the ventral tegmental area (VTA) may contribute to circuit modification induced by many drugs of abuse, including cocaine.
对影响认知的中枢胆碱能神经元的神经营养支持作用、抑制细胞凋亡、调节突触可塑性等可能是它们发挥保护作用的主要机制。
Neurotrophic factors can sustain the function of central cholinergic neurons as well as inhibit the neuronal apoptosis. They can also modulate the synaptic plasticity.
在本论文研究中，我们初步检测了酒精对于小脑突触可塑性的影响，实验结果表明酒精可以影响PF-PC突触LTD的表达。
In this dissertation, our preliminary data showed that alcohol could affect expression of PF-PC synaptic LTD when measuring the influence of alcohol on cerebellar plasticities.
研究人员指出，酒精并不是通过杀死神经元细胞导致失忆发生的。实际上，是这些产生的甾体物质影响了突触可塑性，造成了LTP和记忆形成方面的损害。
The scientists point out that alcohol isn't causing blackouts by killing neurons. Instead, the steroids interfere with synaptic plasticity to impair LTP and memory formation.
通过对出生前有慢性应激经历子代海马CA 1区LTP和LTD诱导性及学习记忆和行为反应的测定，探讨出生前慢性应激对子代海马CA 1区突触可塑性及学习记忆行为的影响。
We explored the effects of prenatal stress on hippocampal synaptic plasticity and behavior of the offspring by testing LTP and LTD in hippocampal CA1 area and learning and memory in Morris Water Maze.
通过对出生前有慢性应激经历子代海马CA 1区LTP和LTD诱导性及学习记忆和行为反应的测定，探讨出生前慢性应激对子代海马CA 1区突触可塑性及学习记忆行为的影响。
We explored the effects of prenatal stress on hippocampal synaptic plasticity and behavior of the offspring by testing LTP and LTD in hippocampal CA1 area and learning and memory in Morris Water Maze.
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