• 各种原因导致基因突变累积相互作用形成基因网络调控的结果,通过干预因子可能达到治疗改变特性的目的。

    Tumors are the results of accumulation of multiple gene mutations and their interactions. So by the intervention on tumorigenic factors, we may cure tumors or change their properties.

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  • 目前治疗胶质细胞疗法直接采用抑制另一种细胞受体上皮生长因子受体(EGFR)。

    Current therapies to treat glioblastomas are directed toward blocking the activity of another cellular receptor called epithelial growth factor receptor (EGFR).

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  • 黑色素确定危险因子之一紫外线所以暴露于日光日光浴可能都是危险因素。

    One of the established risk factors for melanoma is UV light, so both exposure to sun and exposure to tanning beds are probably risk factors.

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  • 目的探讨胰腺癌细胞PC-3胰岛素生长因子-1受体IGF- 1R基因定量表达,及其细胞凋亡、成关系

    Objective To explore Insulin like growth factor 1 receptor(IGF 1R)gene quantitative expression in human pancreatic cancer cells(PC 3) and the relationship of IGF 1R with apoptosis?tumorigenicity.

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  • 目的研究抑制素表皮生长因子受体卵巢颗粒细胞中的表达临床意义

    Objective To investigate the expression of inhibin, epidermal growth factor receptor in human ovarian granulosa cell tumor and their clinical significance.

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  • 目的探讨血管内皮生长因子(VEGF)星形细胞中的表达及其与肿生长、预后血管生成关系

    Objective to study the relationship between the expression of vascular endothelial growth factor (VEGF) and the angiogenesis, growth and prognosis in brain astrocytoma.

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  • 目的研究VX2肝移植微血管密度(MVD)血管内皮生长因子(VEGF)CT灌注成像各灌注参数的相关关系

    Objective to study the relationship of ct perfusion parameters with microvessel density (MVD) and vascular endothelial growth factor (VEGF) in rabbit VX2 liver tumor.

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  • 目的探讨脑膜血管内皮细胞生长因子(VEGF)表达微血管数量(MVQ)良恶性程度的关系

    Objective to investigate the relationship between the expression of vascular endothelial growth factor (VEGF), the microvessel quantity (MVQ) and the grade of human meningiomas.

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  • 结论其他鉴别风险因子算术控制后,转移切除术对转移性黑色素患者有显著生存优势

    CONCLUSIONS: When all other identified risk factors were controlled for mathematically, metastasectomy maintained a significant survival advantage for patients with pulmonary metastatic melanoma.

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  • 目的研究血管内皮生长因子(VEGF)表达人脑转移周脑水肿(PTBE)的关系

    Objective to investigate the expression of vascular endothelial growth factor (VEGF) and its relationship with peritumoral brain edema (PTBE) in brain metastases.

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  • 背景:边缘区淋巴比较少见B细胞淋巴。边缘区淋巴转归预后因子明确

    Background: MZL are uncommon B-cell lymphomas. The outcomes and prognostic factors of MZL remain poorly defined.

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  • 雌激素生理性因子之一逐渐注意到泌乳素形成中的作用

    Estrogen is one of the physiological lactation-promoting factors, and its effects in prolactinoma formation have been gradually noticed.

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  • 目的探讨胶质细胞血小板生长因子B链纯合二聚体(PDGFBB)自分泌活性与其细胞增殖凋亡关系

    Objective to investigate the relationship between activity of autocrine loop of platelet derived growth factor BB (PDGFBB), cell proliferation and apoptosis in gliomas.

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  • 目的研究多发性骨髓MM细胞脑源性神经营养因子BDNF表达水平及其与MM细胞诱导的血管新生关系。

    Objective To investigate the expression of brain-derived neurotrophic factor (BDNF) in multiple myeloma (MM) cells and the correlation between BDNF and MM angiogenesis.

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  • 目的探讨门静脉阻断VX2移植血管内皮生长因子VEGF表达影响。

    Objective To discuss the expression of vascular endothelial growth factor(VEGF)of hepatic VX2 tumor after portal vein occlusion in rabbits.

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  • 探讨晚期神经母细胞(NB)患者血清血管内皮生长因子(VEGF)水平意义

    To investigate the clinical significance of serum vascular endothelial growth factor (VEGF) level in patients with advanced neuroblastoma (NB).

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  • 目的探讨人脑胶质血管内皮细胞生长因子(VEGF)表达微血管数量(MVQ)良恶性程度关系

    Objective to investigate the relationship between the expression of vascular endothelial growth factor (VEGF), the microvessel quantity (MVQ) and the grade in human brain gliomas.

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  • 目的观察血管内皮生长因子(VEGF)抗体大鼠c6胶质细胞凋亡影响对肿生长抑制作用。

    Objective to observe the affect of vascular endothelial growth factor (VEGF) antibody on the cell apoptosis and growth of rat C6 glioma.

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  • 目的探讨应用动态增强磁共振成像(DCE MRI)评价的孤立转移血管通透性血管内皮生长因子(VEGF)表达的空间分布相关性

    Objective to evaluate the correlation between vascular permeability by dynamic-contrast enhanced MRI (DCE-MRI) and vascular endothelial growth factor (VEGF) expression in solitary brain metastasis.

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  • 目的探讨甲状腺转录因子1 (TTF 1)表面活性蛋白a、B (SP A,SP B)硬化性血管(PSH)中表达生物学意义

    Objective to investigate the biologic significance of thyroid transcription factor-1 (TTF-1) and surfactant protein a (SP-A) and SP-B expression in pulmonary sclerosing hemangioma (PSH).

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  • 目的探讨小儿皮肤血管各阶段坏死因子相关凋亡诱导配体(TRAIL)表达细胞增殖凋亡的关系

    Aim: to investigate the relationship between the expression of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL), cell proliferation and apoptosis in children's skin hemangioma.

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  • 目的研究血管内皮生长因子(VEGF)反义rna体外培养脑垂体细胞增殖凋亡影响

    Objective: to study the effect of VEGF antisense RNA on the proliferation activity and apoptosis of human pituitary adenoma cells in vitro.

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  • 目的检测血管内皮生长因子(VEGF)增殖细胞核抗原(pcna)胶质中表达探讨恶性程度关系

    Objective: To examine the expressions of VEGF and PCNA in gliomas, and investigate the possible relationship between their expressions and the degree of tumor malignancy.

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  • 新生血管大量形成实体生长转移中起关键作用血管内皮生长因子(VEGF)介导血管生成的主要因素

    New vessel formation plays a key role in tumor growth and transforming, and the Vascular endothelial growth factor (VEGF) is the most important factor inducing tumor vessel formation.

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  • 目的探讨核糖核酸酶抑制因子胶质生长抑制作用可能机理

    Objective: to explore the role and mechanism of suppression of ribonuclease RNase inhibitor in the growth of glioma cells.

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  • 目的探讨坏死因子相关凋亡诱导体(TRAIL联合化疗药物治疗神经母细胞NB)的作用及其可能机制

    Objective: To explore the role of tumor necrosis factor-related apoptosis-inducing ligand(TRAIL) combined with chemotherapeutic agents in treating neuroblastoma(NB) and the possible mechanism.

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  • 目的探讨用转粒细胞巨噬细胞集落刺激因子(GM CSF)基因苗,联合白介素12 (IL 12)治疗小鼠t细胞淋巴方法

    Objective To study the method of immunotherapy against t cell lymphoma by tumor cells transfected by GM-CSF gene combined with interleukin 12 (IL-12).

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  • 目的研究T细胞细胞游走抑制因子(MIF)基因修饰诱导抗肿免疫反应中的作用

    Objective: To investigate the role of t cell in the antitumor immune responses induced by MIF gene modified tumor vaccine.

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  • 目的研究T细胞细胞游走抑制因子(MIF)基因修饰诱导抗肿免疫反应中的作用

    Objective: To investigate the role of t cell in the antitumor immune responses induced by MIF gene modified tumor vaccine.

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