NKX2-1主要表达于肺组织、甲状腺上皮细胞并广泛分布于前脑腹侧。
NKX2-1 mainly expresses in the lung, thyroid epithelial cells and widely distributes in the ventral forebrain.
临近上皮细胞的胶质内可见清晰空泡，此处上皮组织细胞活跃，甲状腺激素分泌增多，形成扇形的胶质边缘。
Note the clear vacuoles in the colloid next to the epithelium where the increased activity of the epithelium to produce increased thyroid hormone has led to scalloping out of the colloid.
结果甲状腺滤泡上皮细胞平均DNA含量随死后经过时间的延长而加速降解。
Objective To study the relationship between the DNA content in follicular epithelial cells of the human thyroid and postmortem interval(PMI) .
目的探讨甲状腺滤泡上皮细胞平均DNA含量变化与死亡时间（PMI）的相关性。
Methods Changes of the DNA content in thyroid follicular epithelial cells at different PMI were determined by Methyl Green-Pyronin(MGP) stain combined with an image analysis technique.
除髓样癌外，绝大多数甲状腺癌起源于滤泡上皮细胞。
Except for the medullary carcinoma, most of the thyroid carcinoma results from follicular epithelial cells.
CK 18和CK19均是小分子量的角蛋白，含有这些角蛋白的上皮细胞起源不同、分布不同，CK 18 + T Ec和CK 19 +TEc对甲状腺激素的反应性不同。
CK18 and CK19 are cytokeratins with small molecular weights, the CK18 + TECs were different with CK19 + TECs in origin. Distributions and reactions to thyroid hormones.
在电镜下，甲状腺滤泡上皮细胞和腺垂体中的促甲状腺激素细胞的超微结构听见均与其血清中所测激素浓度变化意义相同。
The evidences mentioned above were accord with the morphological changes of thyroid follicular epithelial cell and adenohypophysis thyrotroph seen under electronic microscopy in a sense.
结果，实验组鸡的增重显著高于对照组（P<0.01），其甲状腺滤泡上皮细胞及滤泡腔内胶质均呈现功能活动旺盛的构相。
The results showed that there was significant difference (P<0.01)ir. the body weight between the broilers of group supplied with light and the control group.
观察了饮用高氟水及加硒大鼠的甲状腺组织结构和甲状腺激素变化。结果表明，早期甲状腺滤泡上皮细胞增生活跃、游离甲状腺激素水平显著升高；
Thyroid tissue structure and thyroid hormones of rats with drinking high fluoride(F) water and feeding selenium(Se)feed were observed to study the effects of high F and supplied Se on thyroid.
老年大鼠甲状腺滤泡以及滤泡上皮细胞有显著退行性变化。
The thyroid follicular epithelials in the senile rats degenerated markedly.
老年大鼠甲状腺滤泡以及滤泡上皮细胞有显著退行性变化。
The thyroid follicular epithelials in the senile rats degenerated markedly.
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