这是一个竞争的毒蕈碱型乙酰胆碱受体拮抗剂。
It is a competitive antagonist for the muscarinic acetylcholine receptor.
目的探讨毒蕈中毒的临床表现、治疗与预后关系。
Objective to discuss the relationship of clinical manifestations and treatment to prognosis in mushroom poisoning.
他们发现毒蕈碱能刺激神经兴奋，并引起八目鳗运动。
Their work determined that muscarine stimulated neural activity, leading to locomotion in the laboratory lampreys.
包括直接兴奋毒蕈碱受体的药物和(胆碱酯酶抑制剂)。
Included here are drugs that directly stimulate muscarinic receptors and drugs that potentiate cholinergic activity, usually by slowing the breakdown of acetylcholine (CHOLINESTERASE INHIBITORS).
所的结论为含苷、生物碱、毒蛋白质、毒蕈等成分的食物禁食。
The conclusions of containing glycosides, alkaloids, drugs, proteins, mushroom and other components of food fast.
目的探讨正常幼龄豚鼠眼巩膜组织毒蕈碱受体五种亚型的表达。
Objective to investigate the expression of 5 muscarinic receptor subtypes in the scleral tissue of immature guinea pigs.
这些名字中的每一个都代表文明底下的那些奇形怪状的毒蕈中的一种。
Each one of these names corresponds to a variety of those misshapen fungi from the under side of civilization.
抗胆碱药作用于气道平滑肌上的特定毒蕈碱受体，以抑制迷走张力。
Anticholinergics act by blocking muscarinic receptors in the smooth muscle of the airways and inhibiting vagal tone.
动物性食品是引起中毒的主要食品，毒蕈和农药是中毒死亡的重要原因。
The main food causing poisoning was animal origin and the cause of death was toadstool and pesticides.
对于有这种发病倾向的患者，全身性应用抗毒蕈碱类药物也能诱发青光眼。
Systemic use of the anti-muscarinic agents can also precipitate glaucoma in such predisposed patients.
目的探讨毒蕈碱样乙酰胆碱受体(M受体)在缺氧复合梭曼中毒脑损伤中的作用。
Objective to explore the roles of muscarinic acetylcholine receptor (m receptor) in rat brain injuries after soman intoxication combined with hypoxia.
这些药物，毒蕈碱受体阻断大多被视为有害的副作用，如便秘，镇静，记忆障碍，事业。
With these medications, the muscarinic receptor blockade was mostly viewed as the cause of unwanted side effects, such as constipation, sedation, and memory impairments.
背景：毒蕈碱受体在调节逼尿肌细胞收缩过程中起重要作用，其受体亚型M3R直接介导逼尿肌细胞收缩。
BACKGROUND: Muscarine receptor plays a key role in adjusting contraction of detrusor muscle cell, and M3R, isoforms of its receptor, can mediate contraction of detrusor muscle cell directly.
帕金森病的这些症状可以通过减少脑内乙酰胆碱介导的神经传递而缓解，但很少有人注意到脑干细胞毒蕈碱受体的作用。
Symptoms associated with Parkinson's can be helped by reducing acetylcholine-mediated neurotransmission in the brain, but little work has focused on brainstem muscarine receptors in this disease.
帕金森病的这些症状可以通过减少脑内乙酰胆碱介导的神经传递而缓解，但很少有人注意到脑干细胞毒蕈碱受体的作用。
Symptoms associated with Parkinson's can be helped by reducing acetylcholine-mediated neurotransmission in the brain, but little work has focused on brainstem muscarine receptors in this disease.
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