目的探讨急性弥漫性脑肿胀的发病机制。
Aim To investigate the pathological mechanism of acute diffuse brain swelling.
目的探讨急性弥漫性脑肿胀的发病机理。
Objective?To study the pathologic mechanism of acute diffuse brain swelling.
目的探讨外伤后急性弥漫性脑肿胀的临床特点。
Objective To explore the clinical features of posttraumatic acute diffuse brain swelling (PADBS).
目的探讨外伤后急性弥漫性脑肿胀的诊断和治疗。
Objective To explore the diagnosis and treatment of Posttraumatic Acute Diffuse Brain Swelling (PADBS).
目的探讨外伤后急性弥漫性脑肿胀(PADBS)诊断和治疗。
Objective To explore the diagnosis and treatment of post traumatic acute diffuse brain swelling (PADBS).
探讨小儿外伤性急性弥漫性脑肿胀(PADBS)的发病率及临床特点。
To investigate the morbidity and clinical features of posttraumatic acute diffuse brain swelling (PADBS) in children.
结论外伤后急性弥漫性脑肿胀合并二次脑损伤患者的脑组织超微结构损伤越重,预后就越差。
Conclusions the more severe the ultrastructure is damaged after acute PADBS with SBI, the worse the prognosis of the patients is.
方法对颅脑损伤后出现急性弥漫性脑肿胀的43例病人进行回顾性分析,分析其形成机制及总结治疗措施。
Methods Retrograde analysis of data of post-traumatic acute diffusive edema in 43 cases, mechanism of its development was analyzed and its managements were summarized.
结论大脑血管紧张性调节中枢广泛存在于下丘脑、中脑和延髓,其结构或功能的破坏是急性弥漫性脑肿胀形成的根本基础。
Conclusion there exist vasomotor centers in the hypothalamus, the midbrain and the medulla oblongata, and the destruction of these centers is the basic reason for acute diffuse brain swelling.
目的探讨弥漫性脑肿胀患者术中急性脑膨出的防治措施。
Objective To summarize the prevention and treatment of acute intraoperative encephalocele in patients with diffuse brain swelling.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
结论急性硬膜下血肿,中线移位和脑池受压的程度及有否弥漫性脑肿胀,对判断损伤程度及预后有重大意义。
Conclusion the middle shift, compression of basal cistern and diffuse brain swelling might be helpful in evaluating the severity of craniocerebral injury and the prognosis of patients.
结论急性硬膜下血肿,中线移位和脑池受压的程度及有否弥漫性脑肿胀,对判断损伤程度及预后有重大意义。
Conclusion the middle shift, compression of basal cistern and diffuse brain swelling might be helpful in evaluating the severity of craniocerebral injury and the prognosis of patients.
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