心肌细胞坏死和凋亡的数量与心力衰竭关系明确。
Myocardium apoptosis may provide a new target for heart failure.
毛细血管损伤促进了肌原纤维收缩带形成，加速了心肌细胞坏死。
Injuries of capillaries promoted the formation of contraction band (CB) of myofibril and myocardial necrosis.
根据霍普金斯的看法，这样子的无病毒的细胞可以最终被用来检验新的心脏疾病的药，或用来帮助因心脏病而心肌细胞坏死的病人进行干细胞移植。
Such virus-free cells could eventually be used to test new cardiac drugs, or for stem cell-derived implants to help patients whose cardiomyocytes die in a heart attack, according to Hopkins.
测定心肌病毒滴度、心肌细胞凋亡率和坏死率，计算心肌病理组织学积分。
The virus titer in myocardium, percentage of apoptosis and necrosis of myocytes were detected, myocardial histopathologic scores were counted.
结果表明，VD_3可以引起大鼠心肌细胞膜通透性增大，造成心肌坏死。
The results showed that VD_3 could raise the permeability of rat cardiac sarclemmas and cause cardiac necrosis.
记录心电图的改变，在光镜下观察心肌细胞的炎性浸润和坏死情况，用分离的血清检测心肌酶等。
ECG records the changes in light microscopy myocardial necrosis and infiltration of inflammatory situation, and the separation of serum testing, such as myocardial enzyme.
如果缺血和缺氧是严重的或长期的，心肌细胞就会受到严重的毁灭性的损害和坏死。
If the blood and oxygen supply is cut off severely or for a long period of time, muscle cells of the heart suffer severe and devastating damage and die.
各种心脏疾病发生后，因为心肌细胞增殖能力极低，坏死心肌有纤维组织替代，最终导致心力衰竭。
The necrotic myocardium after various kinds of heart diseases often is replaced by fibrous tissue because of its the very low regenerative potentiality, which will then result in heart failure.
锌能部分地减轻VD_3所致的心肌细胞膜损伤，并对心肌坏死起一定预防作用。
Zinc could partly abate the lesion of rat cardiac sarcolemmas and had certain prophylactic effects on the rat cardiac necrosis.
锌能部分地减轻VD_3所致的心肌细胞膜损伤，并对心肌坏死起一定预防作用。
Zinc could partly abate the lesion of rat cardiac sarcolemmas and had certain prophylactic effects on the rat cardiac necrosis.
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