诱导型一氧化氮合酶的诱导表达往往导致心肌的收缩功能下降。

The up regulation of inducible nitric oxide synthase ( iN OS ) often negatively modulates myocardial function.

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结果发现，陈旧性心肌梗死患者胰岛素抵抗水平和缺失型血管紧张素转化酶基因频率较对照组无统计学差异（P＞0．05）。

Results The insulin resistance level did not increase significantally in patients with old myocardial infarction, compared with control subjects(P>0.05);

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结果发现，陈旧性心肌梗死患者胰岛素抵抗水平和缺失型血管紧张素转化酶基因频率较对照组无统计学差异（P＞0．05）。

Results The insulin resistance level did not increase significantally in patients with old myocardial infarction, compared with control subjects(P>0.05);

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