在增殖期过后第二个三个月里，神经元也被“削减”，这意味着它们经历了一个有计划的细胞死亡过程。
After a period of proliferation during the second trimester, neurons are also "pruned," meaning that they undergo a planned cell death.
区别于杀死目标后很快自身就会死亡的攻击性T细胞，抑制性T细胞可以不断增殖。
And unlike aggressive t cells that die shortly after killing their targets, weak t cells proliferate.
新近研究显示，这一簇miRNA的异常表达可防止癌变前细胞死亡，并令它们增殖成肿瘤。
Recent research has shown that aberrant versions of this cluster are involved in preventing pre-cancerous cells from dying and allowing them to proliferate into tumors.
血管内放射导致细胞死亡的主要形式为凋亡，电离辐射预防再狭窄的主要机制是平滑肌细脑的增殖抑制。
Apoptosis is a major style of cell death induced by endovascular radiation. The inhibition of SMCs proliferation induced by radiation is a main mechanism for prevention of restenosis.
这种缺陷导致单纯疱疹病毒- 1的大量增殖和细胞的死亡。
This deficiency resulted in high rates of herpes simplex virus-1 proliferation and cell death.
结果：经灵芝多糖作用后黑素瘤B16F10细胞增殖活性受抑，细胞死亡增多。
Result: The proliferation of B16F10 cells were inhibited after co-cultured with ganoderma lucidum polysaccharides, and the death proportion of B16F10 cells was increased.
目的探讨死亡受体5 (DR5)对神经细胞增殖的影响。
Objective to investigate the influence of death receptor 5 (DR5) in the proliferation of nerve cells.
目的：探讨细胞增殖与程序性细胞死亡在颞下颌关节发育中的作用。
Objective: To study the role of cell proliferation and programmed cell death(PCD) in the temporomandibular joint(TMJ) development .
结论：细胞增殖与程序性细胞死亡密切相关，协同参与调控颞下颌关节的生长发育与塑形。
Conclusion: Cells proliferation and programmed cell death are interactive with each other in the regulation of the development of TMJ.
线粒体不再被认为是“唯一”作为蜂窝热力学转换器：越来越多的证据牵连他们在细胞信号转导，细胞增殖，衰老，疾病和死亡的重要细胞器。
Mitochondria are no longer considered "only" as the cellular thermodynamic converter: mounting evidence implicates them as key organelles in cell signaling, proliferation, aging, disease and death.
结论临床使用剂量MMC可使结膜囊成纤维细胞增殖能力下降，处于增殖抑制状态，而不引起明显的细胞死亡。
CONCLUSION MMC at clinically relevant doses can inhibit the proliferation of human Tenon's fibroblasts rather than cause the cell death.
细胞分裂中染色体因其末端的DNA不能完全复制而短缩，使细胞逐渐失去增殖能力，导致细胞衰老、死亡。
In cells division chromosome length is shorten for the ending DNA couldn't be replicated completely, and the loss of telomere DNA will lead to senescence and death.
结论：稳定表达人ox40l的乳腺癌转基因细胞在体外能有效地活化t细胞，介导其增殖、分泌细胞因子及抑制T细胞活化诱导的细胞死亡。
CONCLUSION: OX40L-MDA-MB-435 cells could activate t cells in vitro, promote t cell proliferation and cytokine secretion, and suppress t cell activation-induced cell death.
结论 黑龙江立克次体能够感染血管内皮细胞，在血管内皮细胞内不断增殖而使细胞死亡。
Our results demonstrated that R. heilongjiangensis has a capability to infect vascular endothelial cells to cause vascular injury of humans.
结论 黑龙江立克次体能够感染血管内皮细胞，在血管内皮细胞内不断增殖而使细胞死亡。
Our results demonstrated that R. heilongjiangensis has a capability to infect vascular endothelial cells to cause vascular injury of humans.
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