有证据证明IGF - 2在许多肿瘤细胞中可使细胞增殖增加，抑制凋亡，增加局部浸润和转移。
There are evidences that IGF-2 increases cell proliferation, inhibits apoptosis and raises local infiltration and transfer in many tumor cells.
肿瘤的变异分子有许多，如果能阻止1-2个癌细胞的生长就能像试图在某个十字路口放个障碍物就能让全曼哈顿的交通堵塞一样，癌症就可以被抑制生长。
There are so many aberrant molecules in a tumor that blocking just one or two is like trying to stop all traffic in Manhattan with a roadblock at a single intersection.
KIT蛋白在小细胞肺癌中表达，它的激酶活性与许多肿瘤的病理生理学都有联系，包括小细胞肺癌。
KIT protein is expressed in SCLC, and its kinase activity has been implicated in the pathophysiology of many tumours, including SCLC.
结果（1）MIM在许多正常组织细胞中的表达水平明显高于恶性肿瘤细胞。
Results (1) Expressing level of MIM in normal cells were higher than that in tumor metastasis cells.
目的：肿瘤血管生成是肿瘤生长和侵袭的基础，血管内皮细胞生长因子（VEGF）是最重要的血管生成因子，在许多人类肿瘤均可检测到了这种因子。
Objective: Angiogenesis of tumor is the base of tumor growth and invasion. Vascular endothelial growth factor (VEGF) is the most potent angiogenesis factor and has been detected in many human tumours.
骨桥蛋白是一种具有多种功能的磷酸化糖蛋白，不仅广泛分布于骨基质等正常组织细胞中，而且在许多肿瘤组织中都过量表达。
Osteopontin is a multifunctional phosphoglycoprotein. It not only distribute widely in normal tissues such as bone tissues, but also has over expression in multiplicate tumor tissues.
细胞迁移在许多生理和病理过程中都发挥着关键作用，如胚胎发育、神经系统发育、免疫防御、伤口愈合或是肿瘤转移。
Cell migration is involved in a wide variety of physiological and pathophysiological processes, such as embryogenesis, nerve system development, immune defense, wound healing, and tumor metastasis.
FOXP3+CD4+CD25+T细胞是调节机制免疫抵制及免疫无能的一类细胞，在许多人类恶性肿瘤组织中有较高表达，且在肿瘤的发生及进展中起着重要的作用。
It is believed that FOXP3+CD4+CD25+T can regulate the immune suppression and inability, and it is highly expressed in many kinds of malignant tumors.
FOXP3+CD4+CD25+T细胞是调节机制免疫抵制及免疫无能的一类细胞，在许多人类恶性肿瘤组织中有较高表达，且在肿瘤的发生及进展中起着重要的作用。
It is believed that FOXP3+CD4+CD25+T can regulate the immune suppression and inability, and it is highly expressed in many kinds of malignant tumors.
应用推荐