病理学家在突变小鼠之病理学的执行及解读已经扩展并超出传统病理学的范围。
The role of the pathologist in the practice and interpretation of the pathology of the genetically altered mouse has expanded beyond its traditional boundaries.
但研究人员发现，这种重构是在遗传突变小鼠中由于一氧化氮减少而受损，这是与正常小鼠相比较的结果。
But the researchers found that such remodeling was impaired in the mutant mice that produced less nitric oxide when compared to their normal counterparts.
此外，证明突变小鼠比正常小鼠在人类常见的两种疼痛即炎症性疼痛和神经性疼痛方面更敏感。
Further, the mutant mice proved more sensitive than normal mice to inflammatory pain and neuropathic pain, two common forms of chronic pain in humans.
纯合子突变小鼠在各个胚胎时期都有预期中的出现比例，在大小与型态上并没有明显的缺陷。
Homozygous mutant mice showed expected ratio and no obvious defect in sizes and morphologies at all embryonic stages examined.
纯合子突变小鼠在各个胚胎时期都有预期中的出现比例，在大小与型态上并没有明显的缺陷。
Homozygous mutant mice showed expected ratio and no obvious defect in sizes and morphologies at all embryonic stages examined.
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