额外的叶酸，维他命b6和B12能降低大多数病人过高的同型半胱氨酸。
The extra folic acid, vitamins B6 and B12 can lower elevated homocysteine levels in most patients.
B族维生素也极有希望能达到这个目的，因为叶酸、维生素b6、B12能分解同型半胱氨酸——高浓度的同型半胱氨酸也与心脏病有关系。
B vitamins were promising, too, because folate, B6, and B12 help break down the amino acid homocysteine-and high levels of homocysteine have been linked to heart disease.
在维生素b1，维生素b12和叶酸，因为它们都包含在同型半胱氨酸的氨基酸代谢的重要作用。
The vitamins B1, B12 and folic acid are included due to their essential role in the metabolism of the amino acid homocysteine.
他们说，每天0.8毫克的叶酸可以降低同型半胱氨酸浓度，带有和不带有该突变的人群都是。
Folic acid, up to 0.8 mg day, lowers homocysteine concentrations in people with and without this mutation, they say.
而当甲硫氨酸摄入量较低时，同型半胱氨酸又通过一条包含叶酸和维生素b12的途径再循环生成甲硫氨酸。
When methionine intake is low, homocysteine is recycled back to methionine in a process involving folic acid and vitamin B12.
维生素b在那时也大有作为，因为叶酸，B6和B12能帮助摧毁氨基酸同型半胱氨酸——高水平的同型半胱氨酸与心脏病有关联。
B vitamins were promising, too, because folate, B6, and B12 help break down the amino acid homocysteine -and high levels of homocysteine have been linked to heart disease.
这句话没翻译。试译：叶酸治疗组与安慰剂组比较，血清叶酸浓度增加576%且血浆同型半胱氨酸水平下降26%视为治疗有效。
Treatment was effective with serum folate concentrations increasing by 576% and plasma homocysteine falling by 26% for the folic acid group compared with placebo.
尽管已有研究显示同型半胱氨酸与其他心血管疾病之间存在关联，研究人员还没能确定叶酸能否降低除卒中以外的其它疾病的风险。
Despite the link between homocysteine and other cardiovascular diseases, researchers were unable to determine whether folic acid would cut the risk for diseases other than stroke.
结论：低叶酸、维生素b12水平和高同型半胱氨酸水平与随访中抑郁的发生相关，支持单碳物质代谢与抑郁的病因相关。
Conclusion: lower folate, lower vitamin B12 and raised homocysteine levels predicted incident depression, supporting an aetiological role of one-carbon metabolism.
病例与对照之间血浆同型半胱氨酸浓度的差异受到叶酸、维生素B12等因素的影响，而与年龄等因素无关。
The relationship between AD and plasma Hcy level was mainly determined by folate and vitamin B12 levels, but not by age.
病例与对照之间血浆同型半胱氨酸浓度的差异受到叶酸、维生素B12等因素的影响，而与年龄等因素无关。
The relationship between AD and plasma Hcy level was mainly determined by folate and vitamin B12 levels, but not by age.
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