方法：体外培养正常和口腔白斑上皮细胞及成纤维细胞；
Methods: Fibroblast and epithelial cells of normal oral mucosa and OLK were cultured in vitro.
目的：初步探讨口腔白斑的重度上皮异常增生及癌变与吸烟的关系。
Objective: To study the relationship between smoking and malignant transformation of oral leukoplakia.
口腔白斑或纤维化或溃疡、不明皮肤色素淀、体重减轻超过10%以上。
Oral leukoplakia, fibrosis or ulcer, skin pigmentation, loss of weight for over 10%.
结论：端粒酶可以作为口腔白斑癌变化学预防的替代性终点标记物监测口腔白斑癌变。
Conclusion: Ttelomerase could act as a surrogate endpoint biomarker for oral chemoprevention during OLK carcinogenesis.
目的研究口腔白斑(olk)患者病损局部黏膜上皮细胞骨桥蛋白(OPN)表达情况。
Objective To explore the expression of osteopontin (OPN) in oral mucosal epithelium of oral local lesion in patients with oral leukoplakia (OLK).
嚼食槟榔已知为造成口腔白斑、口腔黏膜下纤维化、口腔癌及食道癌之主要危险因子之一。
The evidence of BQ chewing as one of the major risk factors leading to leukoplakia, oral submucous fibrosis, oral cancer and esophagus cancers have been well published.
口腔白斑病是一种相对上较为常见的口腔病灶，其中有少数但显著比例的病例会转变成癌症。
Oral leukoplakia is a relatively common oral lesion that in a small but significant proportion of cases changes into cancer.
方法采用免疫组织化学方法检测17例正常口腔黏膜、60例口腔白斑，30例口腔鳞癌组织标本中TFAR19蛋白的表达。
Methods:The expression of TFAR19 was observed in 17 cases of oral normal mucosa, 60 of oral leukoplakia and 30 of oral squamous cell carcinoma by immunohistochemical stain.
结果提示，口腔白斑组织中egfr的异常表达与口腔白斑上皮异常增和癌变有关，作者认为EGFR的异常表达可能是口腔白斑恶变的较好的标志。
The results suggest that the abnormal expression of EGFR on oral leukoplakia tissue is related to abnormal multiplication and malignant-changes of oral leukoplakia epithelial. The authors...
免疫缺陷的宿主中口腔念珠菌病很普通，例如HIV感染。见毛状舌胎，并有浅棕色渗出物，即毛状白斑舌。
Oral candidiasis is common in immunocompromised hosts, such as those with HIV infection. There is a hairy coating of the tongue seen here mixed with a pale tan exudate.
在114例口腔粘膜病中，以白斑与扁平苔癣居多。
Among 114 cases of oral mucous membrane disease, the leukoplakia and lichen planus are prevalent.
目的：探讨端粒酶基因在口腔黏膜白斑、口腔癌的表达相关性。
Objective: To investigate the relationship of telomerase genes in oral leukoplakia and oral carcinoma.
方法：采用免疫组化染色，光镜及图像分析观察并确定VEGF - C在正常口腔粘膜、白斑及鳞癌等组织中的表达。
Methods: VEGF-C protein expression in benign and malignant oral lesions was investigated with an immunohistochemical staining assay, followed by light microscopic examination and image analysis.
口腔黏膜白斑、背部皮损组织及粪便真菌培养均为白念珠菌生长。
The fungus culture with mucosa blaze in the mouth, lesion on the back and night soil all showed Candida albicans.
方法采用免疫组化之SP_TM法，对20例口腔粘膜白斑，20例口腔鳞状细胞癌和10例正常口腔粘膜进行染色。
Methods By immunohistochSP _TM method, staining 20 cases oral leukoplakia, 20 cases squamous cell carcinoma and 10 cases normal mucosa.
前言：目的：探讨老年口腔黏膜白斑患者的临床特点。
Objectives: To explore the clinical feature of oral leukoplakia in elderly patients.
此外，口腔可出现毛状白斑，毛状白斑的存在是早期诊断艾滋病的重要线索。
In addition, oral hairy leukoplakia can occur, the existence of hairy leukoplakia is an important early clues to diagnosis of AIDS.
有人认为，口腔念珠菌感染和毛状白斑可作为发现或预测艾滋病的指标。
Some people think that oral candidiasis and hairy leukoplakia found or prediction can be used as indicators of AIDS.
结论：氟化处理可降低口腔固定正畸患者釉白斑的发生率。
Conclusion: Fluoride treatments can reduce the incidence rate of enamel opaque spot after orthodontic etching.
有人认为，口腔念珠菌感染和毛状白斑可作为发现或预测艾滋病的指标。
Some people think that oral candidiasis and hairy leukoplakia can be used as an indicator of AIDS found or predicted.
在HIV感染者和艾滋病患者的口腔表征中，卡波济肉瘤的患病率仅次于念珠菌病和毛状白斑，具有较高的发生率。
In HIV-infected people and AIDS patients in the oral representation, the prevalence of Kaposi's sarcoma-rate second only to candidiasis and hairy leukoplakia have a higher incidence.
目的：预防口腔固定正畸酸蚀牙釉白斑的发生。
Objective: To prevent enamel opaque spot after orthodontic etching.
方法通过原位末端转移酶标记法，观察分析10例正常口腔黏膜上皮，18例扁平苔藓，2 3例白斑，2 2例鳞癌上皮组织凋亡状况。
Methods TdT-mediated-dUTP nick end labeling (TUNEL) was used to observe the apoptosis of epithelial cells in 10 cases of normal oral mucosa, 18 cases of LP, 23 cases of LK and 22 cases of SCC.
方法通过原位末端转移酶标记法，观察分析10例正常口腔黏膜上皮，18例扁平苔藓，2 3例白斑，2 2例鳞癌上皮组织凋亡状况。
Methods TdT-mediated-dUTP nick end labeling (TUNEL) was used to observe the apoptosis of epithelial cells in 10 cases of normal oral mucosa, 18 cases of LP, 23 cases of LK and 22 cases of SCC.
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